Journal of Health Economics 37 (2014) 219–231 Contents lists available at ScienceDirect Journal of Health Economics journal homepage: www.elsevier.com/locate/econbase Air pollution and infant mortality: A natural experiment from power plant desulfurization夽 Simon Luechinger ∗ University of Lucerne and KOF Swiss Economic Institute, ETH Zurich, Switzerland a r t i c l e i n f o Article history: Received 28 September 2012 Received in revised form 6 June 2014 Accepted 17 June 2014 Available online 24 June 2014 JEL classification: I12 Q53 J13 a b s t r a c t The paper estimates the effect of SO2 pollution on infant mortality in Germany, 1985–2003. To avoid endogeneity problems, I exploit the natural experiment created by the mandated desulfurization at power plants and power plants’ location and prevailing wind directions, which together determine treatment intensity for counties. Estimates translate into an elasticity of 0.07–0.13 and the observed reduction in pollution implies an annual gain of 826–1460 infant lives. There is no evidence for disproportionate effects on neonatal mortality, but for an increase in the number of infants with comparatively low birth weight and length. © 2014 Elsevier B.V. All rights reserved. Keywords: Health Infants Mortality Infant mortality Air pollution 1. Introduction Health concerns are a primary rationale for air quality regulations such as the U.S. Clean Air Act and the German Federal Immission Control Act. These regulations considerably improved air quality. For example, Fig. 1 depicts the sulfur dioxide (SO2 ) concentration in Germany in 1985–2003. Other developed countries 夽 I thank Wolfgang Bräuniger, Andrea Minkos, and Wolfgang Müller from the German Federal Environmental Agency for the pollution and power plant data, the operating companies for giving confidential information on their generating units, Hiltrud Bayer from the German Youth Institute and the statistical offices of the Länder for the mortality data, and Hans-Peter Mast and Stefan Weil from the Research Data Centers of the Federal Statistical Office and the statistical offices of the Länder for help with remote access to the death and birth certificates. For comments and suggestions, I thank Peter Nilsson, Shinsuke Tanaka, participants of the annual meeting of the Swiss Society of Economics and Statistics 2009, the annual meeting of the European Economic Association 2009, the annual meeting of the American Economic Association 2010, the annual meeting of the German Economic Association 2010 and seminars at the ETH Zurich, the University of Berlin, the University of Fribourg, the University of Mannheim, and the University of Rotterdam as well as the editor Adriana Lleras-Muney and two anonymous referees. ∗ Correspondence to: University of Lucerne, Department of Economics, P.O. Box 4466, 6002 Lucerne, Switzerland. Tel.: +41 41 229 5641. E-mail address: [email protected] http://dx.doi.org/10.1016/j.jhealeco.2014.06.009 0167-6296/© 2014 Elsevier B.V. All rights reserved. experienced similar declines in SO2 concentrations. But this general trend masks considerable heterogeneity. Many people are still exposed to high pollution levels and in developing countries air pollution is often getting worse. Even in Europe, SO2 pollution is bound to rise as coal-based power generation experiences a revival. Therefore, knowledge of the health effects of previous air quality regulations and corresponding improvements in air pollution is of considerable interest. Many studies investigate the effect of air pollution on adult mortality. Epidemiologists typically assess acute effects with timeseries analyses and chronic effects of long-term exposure with cross-section and cohort studies. For example, SO2 was significantly associated with mortality in a time-series analysis for London in 1958–1972 (Schwartz and Marcus, 1990), in a crosssection of groups of U.S. counties in 1970 (Mendelsohn and Orcutt, 1979), or in a large group of adult Americans followed over the period 1982–1998 (Pope et al., 2002). However, studies on infant mortality have clearer implications regarding the number of lifeyears lost and suffer less from uncertainty regarding life-time exposure (Chay and Greenstone, 2003b). Omitted variables are an important concern in all these studies since air pollution depends on economic activity and other unobserved factors with independent effects on mortality. The concern 220 S. Luechinger / Journal of Health Economics 37 (2014) 219–231 Fig. 1. SO2 concentration and infant mortality in East and West Germany, 1985–2003. Sources: Federal Statistical Office and Federal Environmental Agency. is partially addressed in intervention analyses. Evidence of reduced SO2 pollution and mortality following a ban on high sulfur fuel in Hong Kong in 1990 is certainly more convincing than evidence from other time-series analyses, but influences from other concurrent shocks cannot be ruled out without an adequate control group (Hedley et al., 2002). Such concerns sparked a recent interest among economists in natural experiments that allow researchers to identify the effects of air pollution on infant mortality (see Section 2 for a review). This paper estimates the health benefits of an air quality regulation and uses regulation-induced changes in air pollution to identify chronic effects on infant mortality (similar to Chay and Greenstone, 2003a). Specifically, it estimates the effect of the mandated installations of scrubbers at power plants and the resulting reduction in SO2 pollution on infant health with data from Germany in 1985–2003. Thereby, it contributes to the existing literature in two respects. First, the paper provides evidence on infant health effects of air pollution and air quality regulations for another highly developed country than the U.S. Thus, the results help us to understand if and to what extent pollution-mortality relationships found in one context can the transferred to different contexts. While there are no reasons to expect differences in biological dose-response relationships, effects of ambient air pollution also depend on medical care consumption and avoidance behavior, which affects the relationship between ambient air pollution and individual exposure (Graff Zivin and Neidell, 2013). In this regard, Germany and the U.S. may differ along several relevant dimensions. For example, there may be differences in medical care utilization. Pre-natal care can improve infant health and thereby reduce susceptibility to air pollution, good access to medical care after birth allows for timely measures against health problems. Despite a huge increase in Medicaid eligibility of pregnant women in the U.S. in the 1980s, a large share of the population is still uninsured and many eligible women enroll late in their pregnancies (Currie and Gruber, 1996; Gruber, 1997). In contrast, health insurance coverage in Germany is near universal and frequent screenings of infants are statutorily regulated. Similarly, the extent to which individuals can avoid exposure to pollutants may also differ across countries and will depend on such diverse factors as building design with large regional differences in airtightness of houses or typical activity patterns of pregnant women and parents (Ashmore and Dimitroulopoulou, 2009). Second, the paper analyzes the effect of SO2 pollution. Of course, different pollutants may be correlated (Lleras-Muney, 2010) and the regulation may have affected several pollutants. However, as I explain in more detail in Section 3, the German situation analyzed in this paper is particularly well-suited to specifically isolate exogenous variation in SO2 pollution. A federal regulation mandated the installation of scrubbers at power plants and left local authorities or operating companies little room for discretion. Power plants are the main source of SO2 and the predominant scrubbing technology removes SO2 but not other pollutants. Regulation of TSP was already in place and new regulation of nitrogen oxides (NOx ) was generally not binding, affected different sources, and required different compliance measures. Further, I find that the estimated effect of desulfurization at power plants affected SO2 concentration but not NOx concentration. Therefore, I present not only reduced form effects of the policy but also use it to instrument SO2 pollution. Looking at SO2 is interesting for several reasons. First, toxicity differs across pollutants. Second, pollutants differ in the extent to which ambient air pollution translates into individual exposure. For example, the correlation between outdoor and indoor air pollution is lower for SO2 than for other pollutants (Ashmore and Dimitroulopoulou, 2009). Third, the existing evidence on the effects of SO2 on infant mortality is inconclusive and suffers from omitted variables (see Section 2 for a review). Given that SO2 pollution is the focus of many air quality regulations and that there is considerable evidence for effects of SO2 on adult mortality and on adverse pregnancy outcomes (for a review, see Šram et al., 2005), the lack of convincing evidence on the effects of SO2 pollution on infant mortality is regrettable and this paper aims to fill this void. The most important finding is that the air quality regulation had beneficial effects on infant health: Infant mortality decreases with predicted reductions in SO2 concentration due to desulfurization at power plants. The sharp and simultaneous drops in SO2 pollution and in infant mortality between 1987 and 1988 in Fig. 1 anticipates this result. Assuming that the air quality regulation only affected infant mortality through its effect on actual SO2 concentrations, I use the regulation-induced changes in SO2 concentration to estimate the effect of SO2 pollution on infant mortality. According to fixed-effects regressions of infant mortality rates on SO2 concentration, 0.026 infant lives (per 1000 live births) are saved for every 1 g/m3 reduction in SO2 concentration. In instrumental variable regressions with the predicted regulation-induced reductions in SO2 concentrations as an instrument, the effect amounts to 0.045 infant lives. Since most of the variation in SO2 concentration is the result of the air quality regulation, the fixed-effects and instrumental variable estimates are similar and both estimates are informative about the health effects of the regulation. The point estimates translate into an elasticity of 0.07–0.13. The results are similar in subperiods and the West German subsample but not the East German subsample. The estimates are robust to controls for local economic and demographic development, weather, TSP pollution, reunification effects, and rural/urban trends. The instrumental variable estimates are also robust to the inclusion of county-specific time trends, the fixed-effects estimates less so. There is no evidence for strongly disproportionate effects of SO2 on neonatal mortality, but evidence for effects on the number of infants with comparatively low birth weight and, in particular, length. Thus, although poor fetal development due to exposure during gestation seems to affect infant health, it is unlikely to be the main biological mechanisms through which SO2 affects infant mortality. The remainder of the paper is organized as follows. Section 2 briefly reviews the related literature. Section 3 introduces the pollution data and the strategy to instrument SO2 concentrations. S. Luechinger / Journal of Health Economics 37 (2014) 219–231 Section 4 presents the mortality data, the baseline regressions and robustness tests as well as the estimates for mortality for different ages at death and for various birth outcomes. Section 5 concludes. 2. Related literature This paper is closely related to a growing literature in economics that exploits natural experiments to identify the effects of air pollution on infant mortality. Chay and Greenstone (2003a,b) exploit regulation- and recession-induced changes in total suspended particulate (TSP) pollution in U.S. counties in the 1970s and 1980s and find that air pollution increases infant mortality with elasticities between 0.35 and 0.5. Currie and Neidell (2005) and Currie et al. (2009) use within zip-code month variation and find a positive effect of carbon monoxide (CO) with elasticities of 0.09 and 0.04, respectively, but no effects for particulate matter (PM) and ground-level ozone (O3 ). Currie and Schmieder (2009) estimate elasticities for emissions of toxic chemicals ranging from 1.82 for heavy metals to 6.11 for volatile organic compounds and 6.49 for chemicals known to affect child development. Further, there is recent evidence for adverse effects of air pollution on infant survival from developing countries with studies using changes in air pollution due to a voluntary pollution prevention program in Mexico (Foster et al., 2009), wildfires in Indonesia (Jayachandran, 2009), and air quality regulations in China (Tanaka, 2010). In contrast to these well-identified effects of air pollution, the existing evidence on the effects of SO2 on infant mortality suffers from omitted variables. Further, the existing evidence is rather inconclusive. Studies assessing acute effects of SO2 pollution on infant mortality from all causes with time-series analyses consistently find positive effects near a volcano in Japan in 1978–1988 (Shinkura et al., 1999), in Seoul in 1955–1999 (Ha et al., 2003), in São Paulo in 1998–2000 (Lin et al., 2004), and in ten English cities in 1969–1973 (Hajat et al., 2007). The last study estimates an elasticity of 0.03. SO2 pollution has been observed to increase acute deaths from the sudden infant death syndrome (SIDS) in twelve Canadian cities in 1993–2003 (Dales et al., 2004), but not from respiratory causes (Hajat et al., 2007). Results from studies investigating chronic effects from prolonged exposure with crosssection, pooled panel, or case–control studies generally discover no significant effects of SO2 on all-cause infant mortality. This is the case for studies using data across U.S. standard metropolitan statistical areas in 1961–1964 (Hickey et al., 1976), U.S. county groups in 1970 (Medelsohn and Orcutt 1979), counties and boroughs in England and Wales in 1969–1973 (Chinn et al., 1981), Czech districts in 1986–1993 and in 1989–1991 (Bobak and Leon, 1992; 1999), or large U.S. counties in 1999–2002 (Woodruff et al., 2008). An exception is the study by Joyce et al. (1989) with data from U.S. counties in 1970, which finds SO2 pollution to increase neonatal mortality with an elasticity of 0.02–0.05. The results from studies researching the effects of prolonged exposure on mortality from specific causes are conflicting. Chinn et al. (1981) and Woodruff et al. (2008) find no effect on pneumonia or respiratory mortality, while Bobak and Leon (1992; 1999) find positive effects on respiratory mortality. Lipfert et al. (2000) and Woodruff et al. (2008) discover no effect on SIDS mortality. Crocker et al. (1979) estimate mortality from early infant diseases to increase with SO2 in a cross-section of 60 U.S. cities in 1970 with an elasticity of 0.09. 3. Pollution: data, evolution and instrument From the German Federal Environmental Agency (Umweltbundesamt; hereafter UBA for short) I have data on the annual mean SO2 221 concentration measured at air quality monitors for 1985–2003.1,2 There are data for between 196 monitors in 1985 and 416 monitors in 1994, or 553 monitors in total. I interpolate the monitor readings on a grid with cells of 1 km2 . For the interpolation, I use the method of inverse distance weighting with the 9 nearest monitors (without any distance cutoff) and the inverse cubed distance as weights. The UBA determined the parameters on the basis of empirical studies. However, both interpolated values and regression results are very similar for slightly different parameters.3 Following the approach suggested in Currie and Neidell (2005), I evaluate the accuracy of the interpolation procedure by comparing at each monitor actual readings with the concentration level that would be estimated with the interpolation procedure if this particular monitor was not there. The correlation of 0.87 implies that the interpolated values are accurate. The results are very similar if the sample is reduced to county-years with active monitors.4 The number of monitors changes over time and the placement of the monitors may be endogenous. However, if I use pollution measures based on the 64 continuously operating monitors, the results get even stronger but are broadly similar.5 To merge the air pollution data with the mortality data, I aggregate the interpolated pollution values to the county level by taking the average value of all grid cells that fall within the borders of a county.6 Fig. 2 depicts the mean SO2 concentration per county in 1985, 1990, 1995, and 2000. Looking at the pattern and evolution of SO2 pollution, two aspects are worth noting. First, in the mid-1980s, pollution levels were high, especially at three hotspots, the Ruhr area in the west, Northern Hesse in the center, and the area around Leipzig in the east. Back then, these areas were important industrial centers and coal mining areas. Second, air quality diminished substantially after 1985 and 1990 in West Germany and after 1990 in East Germany. These improvements are largely the result of the large combustion plant ordinance enacted in 1983. The ordinance required operating companies to retrofit fossil fuel fired power plants with scrubbers within three to nine years from 1986 on. Retrofitting deadlines were statutorily fixed and depended on a plant’s capacity and emissions. Thus, they were not chosen by regulators or operating companies. The unification treaty of 1990 extended the regulation to East Germany with East German power plants required to install 1 The pollution data and identification strategy have been previously used in Luechinger (2009) to estimate the effect of air pollution on subjective well-being. The description of the data and identification strategy in this section is based on the description of Luechinger (2009). 2 The UBA calculates annual mean concentrations as simple averages based on daily or hourly means. The temporal aggregation follows EU rules which require minimum data capture of 50 percent for annual means (2001/752/EC and email from Andrea Minkos, UBA, June 14, 2013). 3 The baseline estimates reported in column 1 of Table 2 for the cubed distance and the nine nearest monitors are 0.026 (std. err.: 0.004) (fixed-effects estimate) and 0.044 (std. err.: 0.009) (instrumental variable estimate). Using the squared distance and the six nearest monitors, the respective estimates are 0.026 (std. err.: 0.004) and 0.044 (std. err.: 0.009), using the cubed distance and the six nearest monitors 0.030 (std. err.: 0.005) and 0.046 (std. err. 0.010) and using the squared distance and nine nearest monitors 0.027 (std. err.: 0.004) and 0.044 (std. err. 0.009). 4 The baseline estimates reported in column 1 of Table 2 are 0.026 (std. err.: 0.004) (fixed-effects estimate) and 0.044 (std. err.: 0.009) (instrumental variable estimate). For the reduced sample the respective estimates are 0.021 (std. err.: 0.006) and 0.039 (std. err.: 0.012). Thus, despite of a reduction of the sample size by nearly 60 percent, the results are quantitatively similar and still precisely estimated. 5 The baseline estimates reported in column 1 of Table 2 are 0.026 (std. err.: 0.004) (fixed-effects estimate) and 0.044 (std. err.: 0.009) (instrumental variable estimate). The respective estimates with pollution measures based on the 64 continuously operating monitors are 0.046 (std. err.: 0.009) and 0.064 (std. err.: 0.014). 6 County mergers in East Germany caused the number of counties to fall from 543 in 1993 to 439 in 2001. The analysis in this paper is based on the 439 counties at the end of the merging process. 222 S. Luechinger / Journal of Health Economics 37 (2014) 219–231 the product of the operating status, pre-desulfurization or residual emissions, a distance decay function, f(Dcj ), and the frequency county c lies downwind of power plant j, g(Rcj ). The contribution from all power plants is the sum of the contributions from individual plants multiplied by an unknown parameter ˛1 converting pollutant mass into concentration levels. Thus, the SO2 concentration in county c at time t can be described by Eq. (1): Pct = ˛0 + ˛1 1(active)jt · Ej · (1 − ˛2 · 1(scrubber)jt ) j ·f (Dcj ) · g(Rcj ) + c + t + εct . (1) Slightly re-arranging terms yields Eq. (2): Pct = ˛0 + ˛1 −˛1 ˛2 1(active)jt · Ej · f (Dcj ) · g(Rcj ) j (2) 1(active)jt · Ej · 1(scrubber)jt · f (Dcj ) · g(Rcj ) + c + t + εct . j The third term in Eq. (2), 1(active)jt · Ej · 1(scrubber)jt · f (Dcj ) · j g(Rcj ), is the estimated effect of desulfurization at power plants on SO2 pollution, which I will use to identify the effects of air pollution. The temporal variation comes from two sources: The retrofitting of power plants with scrubbers and changes in the power plant population. Only the first is exogenous. Changes in power plant population may be related to unobserved factors with independent effects on infant mortality. Therefore, I include the effect of changes in the power plant population captured by the second term in Eq. 1(active)jt · Ej · f (Dcj ) · g(Rcj ), as a control variable. The geo- (2), j Fig. 2. SO2 concentration in German counties; 1985, 1990, 1995 and 2000. Legend: ≤ 20 g/m3 , 20–40 g/m3 , 40–60 g/m3 , 60–80 g/m3 , 80–100 g/m3 , 100–125 g/m3 , 125–150 g/m3 and > 150 g/m3 ; cities: D = Dortmund in the Ruhr area, K = Kassel in Northern Hesse, L = Leipzig and B = Berlin. Sources: See text. scrubbers from 1993 on. Again, different retrofitting deadlines for different categories of power plants were statutorily fixed. Below I will use the estimated effect of the mandated installation of scrubbers at power plants to identify the effects of air pollution on infant mortality. This effect is estimated with a simple model of air pollution, information on power plants’ operating status, annual pre-desulfurization SO2 emissions, and retrofitting status, and information on wind directions as well as distances and directions between power plants and counties. The SO2 concentration in county c at time t, Pct , comprises contributions from power plant emissions and background pollution. Background pollution is captured by county effects, c , time effects, t , and a random component, εct . The contribution of emissions from an individual plant j depends on the operating status and the retrofitting status of the power plant, 1(active)jt and 1(scrubber)jt , pre-desulfurization emissions or residual emissions, Ej or Ej · (1 − ˛2 · 1(scrubber)jt ), and the distance and direction between the plant and the county, Dcj and Rcj . 1(active)jt is a dummy with value one if a power plant operates at time t and zero otherwise, 1(scrubber)jt is a dummy with value one if the power plant has installed a scrubber at time t and zero otherwise, and ˛2 is an unknown parameter reflecting average separation efficiency of scrubbers. The contribution of an individual power plant j is graphical variation also comes from two sources: Distance to power plants and the distribution of wind directions. The estimated effect of the installations of scrubbers on air pollution is the interaction of temporally and geographically varying components and, thus, similar to a difference-in-difference term. However, the term differs from a standard difference-in-difference term in three respects. First, treatment and control group status is a matter of degree rather than one of kind and depends on distance and wind direction frequencies. Second, I have to include all power plants simultaneously. Therefore, my treatment variable is a weighted sum of desulfurization at all plants with predesulfurization emissions as weights. Third, some power plants are newly constructed, others taken offline. For this reason, I control for changes in the power plant population. For over 300 fossil fuel fired generating units with a capacity of 100 MW or more and active between 1985 and 2003, I have information on the starting year, the year the unit was taken offline, the year of desulfurization, capacity, fuel and fuel efficiency. The information comes from the UBA, publications of operating companies and the engineering literature, a questionnaire sent to operating companies, and statutory provisions (see Luechinger, 2009 for details). Panel A of Fig. 3 pictures power plants’ locations. I use published emission factors (Bakkum et al., 1987) and plants physical characteristics to estimate the pre-desulfurization SO2 emissions.7 7 Lacking data on utilization rates, I have to assume full utilization of capacities. The emissions are then simply the product of the emission factor, the capacity, inverse fuel efficiency, and the time period. This calculation may overstate emissions because the assumption of full utilization may not be plausible. However, for my purpose, the absolute level of emissions is immaterial. I am only interested in the relative size of pre-desulfurization emissions from different power plants, which are mainly due to differences in fuels and plant size. Further, using actual S. Luechinger / Journal of Health Economics 37 (2014) 219–231 Fig. 3. Locations of fossil fuel fired power plants and wind stations. Sources: See text. I model distance decay with an exponential curve and a characteristic decay distance of 480 km, i.e. g(Dcj ) = exp(−2.1E-6 · Dcj ), as proposed by field estimates (Schwartz, 1989; Summers and Fricke, 1989). Frequencies of wind directions in 12 30-degree sectors at the nearest wind station characterize the wind situation at each power plant (Traup and Kruse, 1996). Panel B of Fig. 3 depicts the 43 wind stations used in this paper. Finally, I use the distance and direction between every power plant and every county to relate plant- and county-level data. To interpret the effect of the mandated installations of scrubbers at power plants as the causal effect of improved air quality, I need to assume that the retrofitting of power plants affected infant health only through its effect on air quality. In this context, it is important to note that the statutory provisions were enacted prior to the sample period. Thus, it is unlikely that the actual installation of scrubbers is a response to concurrent demographic, economic or political developments in faraway upwind or nearby downwind regions. However, one might worry that counties lying downwind in the vicinity of a retrofitted power plant experienced improvements in air quality and infant health before the plant installed the scrubber. In this case, the difference-in-difference term in Eq. (2) that is used as an instrument would partly capture these preexisting trends. In order to assess this issue, Fig. 4, panels A and C, plots for each county the differences in SO2 concentration and infant mortality between the first year of the desulfurization process and the last year before the process against the estimated effect of desulfurization averaged over the whole sample period. The predesulfurization differences relate to the years 1986–1985 for West Germany and the years 1993–1992 for East Germany. Fig. 4, panels B and D, also plots the differences in SO2 concentration and infant mortality between the third and the first year of desulfurization process against averaged values of the estimated effect of desulfurization. The post-desulfurization differences relate to the years 1988–1986 for West Germany and the years 1995–1993 for East Germany. These years mark the time frame during which the most polluting power plants were required to install scrubbers. Fig. 4 plots both actual values and Kernel-weighted local polynomial regression-smoothed values. As we can see from Fig. 4, in the years before the desulfurization process, average values of the estimated effect of desulfurization utilization rates may be problematic because these rates are endogenous and potentially related to unobserved factors. 223 are not related to changes in SO2 concentration. The correlation () is −0.05 (p = 0.294). In contrast, average values of the estimated effect of desulfurization are strongly related to changes in SO2 concentration after the desulfurization process started ( = −0.49; p < 0.0001). The same pattern holds for infant mortality rates even though it may be less visible to the naked eye. The correlation for pre-desulfurization is 0.05 (p = 0.276), post-desulfurization it is −0.11 (p = 0.018).8 Another worry might be that that the retrofitting of power plants lowered concentrations of other pollutants in addition to SO2 . However, the German situation analyzed in this paper is wellsuited to specifically isolate exogenous variation in SO2 pollution. Air quality regulation in other countries, notably the U.S., often leaves authorities at subnational levels substantial leeway in formulating their own implementation plans and in deciding how pollution targets are reached. Similarly, changes in economic activity are likely to affect several pollutants. In contrast, the large combustion plant ordinance specified not only targets but also measures and it left local authorities or operating companies little room for discretion. The regulation of TSP had a long tradition in Germany before the large combustion plant ordinance. However, in addition to emission limits for SO2 , the large combustion plant ordinance did establish for the first time emission limits for nitrogen oxides (NOx ), though the limits were preliminary and generally not binding. Further, for four reasons my instrument is unlikely to capture changes in NOx pollution. First, power plants are the most important source of SO2 , but not of NOx pollution. For example, in 1990 power plants accounted for 60 percent of all SO2 emissions but only for 20 percent of NOx emissions; NOx emissions are primarily caused by road traffic (UBA, 2012). Second, SO2 emission factors of coal and oil fired power plants are two to five times as large as NOx emission factors. Conversely, gas fired power plants are important emitters of NOx , but not SO2 (Bakkum et al., 1987). Third, 93 percent of the installed scrubber capacity was wet scrubbers with a water-gypsum-limestone/lime-slurry as scrubbing liquid (Mittelbach, 1991). This scrubbing technology is aimed at removing SO2 , not NOx . Fourth, and most importantly, for the years 1990–2003, the years for which NOx data are available, there is a strong first stage for SO2 (t-value: −4.29) but none for NOx (t-value: 0.45). 4. Effect of SO2 pollution on infant mortality 4.1. Data and empirical strategy The state statistical agencies collect data on births and deaths in standardized way in accordance with federal laws and then publish infant mortality rates at the county level in state reports or make them available upon request. The German Youth Institute, an independent children and family research institute, compiled the data for years since 1986 and generously shared the data with me. The 1985 data come directly from the statistical agencies.9 Infant 8 It is important to note that the actual development stacks the deck against finding the instrument to be valid. Even though power plants were not statutorily required to install scrubbers before 1986 and 1993 in West and East Germany, respectively, individual power plants (<6 percent) were retrofitted before these dates to test desulfurization technologies or in connection with regular overhauls. 9 Data on infant mortality are available for 326 Western counties and West Berlin/Berlin for 19 years (1985–2003) and for 112 Eastern counties over 14 years (1990–2003) or for a maximum of 7781 county-years. I drop observations when the infant mortality rates are decreasing with increasing life span, e.g. if the mortality rate for deaths within the first 28 days is larger than the mortality rate for deaths within the first year, because this is indicative of coding errors. This reduces the number of observations by 73. Further, 3 observations for one county are 224 S. Luechinger / Journal of Health Economics 37 (2014) 219–231 A. Pre-desulfurization pollution differences B. Post-desulfurization pollution differences 20 20 0 0 -20 -20 -40 -40 -60 -60 Average estimated effect of desulfurization per county C. Pre-desulfurization mortality differences Average estimated effect of desulfurization per county D. Post-desulfurization mortality differences 20 20 10 10 0 0 -10 -10 -20 -20 Average estimated effect of desulfurization per county Average estimated effect of desulfurization per county Fig. 4. Pre- and post-desulfurization pollution and infant mortality differences and average estimated effect of desulfurization per county. Notes: (1) Pre-desulfurization differences relate to the years 1986–1985 for West Germany and the years 1993–1992 for East Germany, post-desulfurization differences to the years 1988–1986 for West Germany and 1995–1993 for East Germany. (2) The panels show the actual and Kernel-weighted local polynomial regression-smoothed values. Stata’s default options are used for Kernel smoothing, i.e. an Epanechnikov kernel function, zero degree polynomial and rule-of-thumb bandwidth. mortality is reported as the number of deaths within the first year per 1000 live births. According to the summary statistics in Table 1, 5.76 infants per 1000 live births died on average in the sample period. For the years 1986–1999, I have disaggregated data on the number of deaths within 1 day and 28 days. Due to changes in federal laws, states no longer reported detailed information on early infant deaths in later years. For the analysis on disaggregated mortality rates and birth outcomes, I also use microdata stemming from the death and birth certificates for the years 1991–2003.10 I combine the birth and the mortality data by aggregating both to the level of county × year × sex × legitimacy-cells. For confidentiality reasons, the microdata are hosted at the Research Data Centers of the Federal Statistical Office and the statistical offices of the Länder and have to be analyzed by remote access. In 1991–2003, 5.33 infants died on average per 1000 live births. The explanatory variable of interest is the annual mean concentration of SO2 introduced in Section 3. The mean concentration missing in the original data. 117 observations for Eastern counties (incl. Berlin) before 1991 are dropped because of missing control variables. However, dropping these 117 observations has little effect on the results. The baseline estimates reported in column 1 of Table 2 are 0.026 (std. err.: 0.004) (fixed-effects estimate) and 0.044 (std. err.: 0.009) (instrumental variable estimate). For the larger sample with 7705 observations, the respective estimates are 0.023 (std. err.: 0.003) and 0.041 (std. err.: 0.008). The original data refer to infant mortality rates in counties that existed at that particular point in time. I account for county mergers in East Germany (see footnote 8) by taking simple averages of merging counties since I lack data on births and since counties did not always merge integrally. 10 Microdata for Saarland in 1991 and Mecklenburg-Vorpommern in 1991–1994 are missing. is 16.03 g/m3 in the 1985–2003 sample and 12.18 g/m3 in the 1991–2003 sample. A first set of control variables includes basic economic and demographic variables, namely GDP per capita, employment and population. The data are from Cambridge Econometrics. A second set of control variables includes weather variables. Weather and, in particular, extreme weather conditions affect both pollution and mortality (Deschênes and Greenstone, 2011). Therefore, I control for the mean temperature in the coldest month, the mean temperature in the hottest month, the mean precipitation in the driest month, and the mean precipitation in the wettest month in some specifications. To construct these variables, I use for each county daily data from the nearest of 35 weather stations from the German meteorological office (Deutscher Wetterdienst) or the European Climate Assessment and Data with continuous readings. In the extensions with the microdata aggregated to the county × year × sex × legitimacy-level, I control for sex and legitimacy status of infants as well as for three characteristics of mothers at the cell-level: the average age of mothers, the percentage of mothers with German citizenship, and the percentage of working mothers. In the following, I estimate different variants of the following empirical model: IMRct = ˇ0 + ˇ1 Pct + ˇ2 Zct + c + t + εct , (3) where IMRct is the infant mortality rate in county c in year t, Pct the SO2 concentration in this county and year, Zct a vector of control variables, c and t county and year effects and εct an error term. The relationship between pollution and mortality is modeled linearly since F-tests reject models with second or third order polynomials in the present case. S. Luechinger / Journal of Health Economics 37 (2014) 219–231 225 Table 1 Summary statistics. Obs A. County × year-level data, 1985–2003 Infant mortality rate (w/in 1 year; per 1000 live births) SO2 (g/m3 ) B. County × year-level data, 1986–1999 Infant mortality rate (w/in 1 year; per 1000 live births) Infant mortality rate (w/in 1 day; per 1000 live births) Infant mortality rate (w/in 28 days; per 1000 live births) SO2 (g/m3 ) C. County × year × sex × legitimacy-level data, 1991–2003 Infant mortality rate (w/in 1 year; per 1000 live births) Infant mortality rate (w/in 1 day; per 1000 live births) Infant mortality rate (w/in 28 days; per 1000 live births) Stillbirths (per 1000 births) Weight Small for gestational age, weight (per 1000 live births) Length Small for gestational age, length (per 1000 live births) SO2 (g/m3 ) Mean Median Std. Dev. Min Max 7588 7588 5.76 16.03 5.40 10.50 2.91 16.13 0.00 1.54 23.30 174.07 5481 5481 5481 5481 6.03 1.45 3.35 18.14 5.70 1.30 3.10 13.10 2.86 1.25 1.99 16.03 0.00 0.00 0.00 1.67 23.30 9.40 20.40 174.07 22,500 22,500 22,500 22,500 22,500 22,500 22,500 22,500 22,500 5.33 1.44 3.10 4.37 3331 36.45 50.98 34.34 12.18 4.01 0.00 1.07 2.98 3330 33.71 51.02 30.97 7.52 6.67 3.30 4.95 5.79 102 18.11 0.69 18.77 15.08 0.00 0.00 0.00 0.00 2876 0.00 47.73 0.00 1.54 142.86 48.78 83.33 114.29 3756 208.33 53.56 193.55 174.07 Sources: (1) German Youth Institute; statistical offices of the Länder; (2) Research Data Centres of the Federal Statistical Office and the statistical offices of the Länder, statistics of births and statistics of deaths, 1991–2003; (3) Federal Environmental Agency; own analysis. Notes: Small for gestational age is here defined as birth weight or length more than two standard deviations below the mean of the birth cohort. To account for serial and spatial correlations, I cluster standard errors at the level of 92 analytical regions defined by the Federal Office for Building and Regional Planning. These regions, determined on the basis of commuter flows, encompass an economic center and its periphery and are often congruent with sates’ planning regions. Thus, it is likely that counties within these regions are exposed to common shocks. 4.2. Basic results Table 2 presents the basic results for the whole sample. It presents the results from OLS (panel A), fixed-effects (panel B), and instrumental variable (panel E) regressions as well as the reduced form (panel C) and first stage (panel D) related to the instrumental variable regressions. All estimates are intended to assess the health benefits of the large combustion plant ordinance, which accounts for a large part of the reduction in SO2 pollution in Germany. Though most of the variation in SO2 pollution is the result of this specific regulation, highly polluted counties and counties with large regulation-induced improvements in air quality will differ from other counties along several, potentially unobservable dimensions. Fixed-effects in panels B-E account for this cross-sectional heterogeneity across counties. In addition, the instrumental variable estimates correct for classical measurement errors and for unobserved factors that may have accompanied non-regulationinduced changes in pollution. Table 2 presents the results for three different specifications: Column 1 presents estimates with only year effects as controls, column 2 adds economic variables, and column 3 weather conditions. The results reported in Table 2 suggest that infant mortality increases with air pollution. According to the OLS estimates, 0.032 infant lives are lost per 1000 live births for every 1 g/m3 increase in SO2 concentration (column 3). Accounting for cross-sectional heterogeneity by including fixed-effects only marginally affects the results. In column 3, the point estimate is slightly reduced to 0.026 infant deaths per 1000 live births. Using the exogenous variation introduced by the large combustion plant ordinance, we can see that infant mortality decreases with increasing reductions in air pollution due to flue gas desulfurization at power plants. Thus, the environmental regulation had the intended health effects. The first stage results show that actual SO2 concentration is lower, the larger the predicted reduction in SO2 concentration is. As explained in Section 3, the respective variable is akin to a difference-in-difference term with the temporal variation coming from the staggered installation of scrubbers at power plants and the geographical variation from the distance and direction between a county and power plants, whereby the direction determines the frequency the county lies downwind of the plants. If I construct a similar term without taking wind frequency into account, the resulting instrumental variable estimates are very similar to the ones reported in Table 2 (differences of less than 6 percent). Thus, distance to power plants seems to be more important for the differential effects of desulfurization across counties than direction. The size of the reduced form coefficients is difficult to interpret. If I scale the coefficients by the respective first stage estimates, the resulting instrumental variable estimates imply a marginal effect of SO2 on infant mortality of 0.045 (column 3). Thus, the instrumental variable estimates are slightly larger, but the difference is small and Hausman tests reject the null hypotheses of equality of fixedeffects and instrumental variable estimates at the 5 percent level at best. This is consistent with the notion that most of the variation in pollution is the result of the large combustion plant ordinance. There are several ways to put the results into perspective. In the following, I will compute the implied elasticity and compare them to previous estimates, calculate the number of infant lives saved by the improvement in air quality, estimate the contribution of improvements in air quality to the overall decline in infant mortality rates, and monetize the reductions in lost infant lives. The marginal effects of 0.026–0.045 translate into an elasticity in the range of 0.07–0.13, which is at the lower end of the elasticities reported in the recent economics literature. It is lower than the elasticities for chronic effects of toxic chemicals of 1.82–6.49 (Currie and Schmieder, 2009) and for chronic effects of TSP of 0.35–0.5 (Chay and Greenstone, 2003a,b), but larger than the elasticities for acute effects of CO of 0.04–0.09 (Currie and Neidell, 2005; Currie et al., 2009). It is larger than an estimated elasticity for acute effects of SO2 of 0.03 (Hajat et al., 2007). There are several reasons for these differences. First, pollutants differ in their toxicity and the degree to which exposure to them can be avoided. The wide range of elasticities reported by Currie and Schmieder (2009) for different toxic chemicals nicely illustrates this point. Second, acute effects may differ from chronic effects. For example, chronic effects of particulate pollution on infant mortality are generally larger than acute effects (Lacasaña et al., 2005). Finally, as 226 S. Luechinger / Journal of Health Economics 37 (2014) 219–231 Table 2 Basic results: Effect of SO2 pollution on infant mortality, 1985–2003. (1) (2) (3) 0.029** (0.005) 0.25 0.030** (0.005) 0.25 0.032** (0.005) 0.26 0.026** (0.004) 0.27 0.026** (0.004) 0.27 0.026** (0.004) 0.27 −0.224** (0.051) 0.26 −0.217** (0.051) 0.26 −0.226** (0.052) 0.27 −5.135** (0.502) 0.72 104.49 −4.949** (0.468) 0.73 111.89 −4.995** (0.484) 0.74 106.39 R2 Hausman test p-value 0.044** (0.009) 0.27 0.073 0.044** (0.009) 0.27 0.068 0.045** (0.009) 0.27 0.047 Control variables Year effects Economic variables Climate variables No. of observations No. of counties No. of clusters Y N N 7588 439 92 Y Y N 7588 439 92 Y Y Y 7588 439 92 A. OLS estimates Dependent variable Infant mortality rate Pollution SO2 R2 B. Fixed-effects estimates Dependent variable Infant mortality rate Pollution SO2 R2 C. Reduced form estimates Dependent variable Infant mortality rate Pollution Predicted SO2 R2 D. First stage estimates Dependent variable SO2 Excluded instrument Predicted SO2 R2 F-stat excl. instrument E. IV estimates Dependent variable Infant mortality rate Pollution SO2 Notes: (1) Panel A reports OLS regressions; panels B-E report regressions with county fixed effects. The instrumental variable is the estimated reduction in SO2 pollution due to desulfurization at power plants for each county-year. It is estimated by summing over all power plants the product of a dummy variable with value one if a power plant operates at time t and zero otherwise, estimated pre-desulfurization emissions, a dummy variable with value one if a power plant has installed a scrubber at time t and zero otherwise, a distance decay function, and the frequency a county lies downwind of a power plant. The instrumental variable is included together with a control variable capturing the estimated effect of changes in the power plant population on SO2 pollution. It is estimated in a similar way as the instrumental variable but without the dummy variable for the installation of scrubbers. (2) The unit of observation is the county-year. (3) Cluster robust standard errors in parentheses allow for clustering at the level of 92 analytical regions encompassing several counties. (4) ** is significant at the 99 percent level. discussed in the introduction, differences across countries regarding health care utilization and many other aspects may influence the relationship between air pollution and infant mortality. The average decrease in SO2 concentration in counties between the first and the last year that they are in the sample is around 43 g/m3 . With around 746,000 live births each in year in Germany, the number of infant lives saved amounts to around 826 for the fixed-effects estimates and to around 1460 for the instrumental variable estimates. The average decrease in infant mortality in counties over the sample period is 4.40 per 1000 live births. Thus, by multiplying the average reduction in SO2 pollution of 43 g/m3 with the coefficients in column 3 and then relating the resulting product to the average decrease in infant mortality rates, I find that between 25 and 44 percent of the decrease in infant mortality over the sample period is due to the improvement in air quality. If we are prepared to monetize these benefits in terms of infant health, we can compare them to the costs of regulation. Chay and Greenstone (2003a,b) use a value of a statistical life (VSL) estimate of $1.7 million (2000 US$) based on a study by Ashenfelter and Greenstone (2004); Currie and Neidell (2005) use EPA’s estimate of $5.0 million (2000 US$); the median VSL estimate for prime aged workers in the U.S. in the meta-analysis of Viscusi and Aldy (2003) is $6.7 million (2000 US$). Hence, the estimates differ by an order of magnitude and so will the estimates of infant health benefits. The lowest annual benefit estimate for each of the around 27,793,000 households (and 727,199 births) in the year 1989/1990 in West Germany based on the fixed-effects estimate and the VSL estimate of Ashenfelter and Greenstone (2004) is around $50 (2000 US$), the highest estimate based on the instrumental variable estimate and the VSL estimate of Viscusi and Aldy (2003) is around $343 (2000 US$). These benefit estimates compare favorably to rough compliance cost estimates for West Germany in the range of between $33 and $165 per year and household (2000 US$).11 Of course, the large combustion plant ordinance is not the sole reason for improvements in air quality. At the same time, the reduction in infant mortality may well reflect much broader health benefits. Further, a potentially important cost of air pollution not reflected in the estimates are the defensive steps individuals take in order to avoid exposure to air pollution or adverse health effects (Bresnahan et al., 1997; Neidell, 2009; Graff Zivin and Neidell, 2009; Moretti and Neidell, 2011; Deschênes et al., 2012). Thus, it is very likely that overall benefits of cleaner air exceed the health benefits presented here. 4.3. Robustness tests The analysis so far has been based on the annual mean SO2 concentration in counties. In the present context, this is the appropriate pollution measure for three reasons. First, I am interested in chronic effects resulting from prolonged exposure to air pollution, not in acute effects to extreme events. Second, the temporal resolution of the infant mortality data is inherently annual. Third, lacking temporally more disaggregated data on power plants, the instrumental variable also has an annual resolution. Nevertheless, I also estimate the effects for three alternative measures of SO2 concentration, the annual maximum concentration and the number of days the German and the former U.S. 24-h air quality standards of 125 g/m3 and 365 g/m3 are exceeded.12 With the exception for the 11 Schärer and Haug (1990) estimate costs of the desulfurization at West German power plants at DM 14.2 billion (1988 DM). I double this value to crudely consider operating costs, assume a real long-term interest rate of 5 percent, and divide by 27,793,000 households (living in Germany in 1989). This results in costs of $33 (2000 US$) per household and year. At the other end, Schulz’ (1985) most pessimistic cost forecast is DM 9 per person and month (1984 DM assumed) or $165 (2000 US$) per household and year. 12 Germany has since 2002 ambient air pollution standards at the federal level. The 24-h SO2 standard of 125 g/m3 is not to be exceeded more than three times a year (22nd BImSchV, September 11, 2002). In the U.S. in 1971–2010, the primary SO2 standard was the 24-hour standard of 365 g/m3 , which was not to be exceeded more than once per year (see, e.g., Federal Register, “National ambient air quality standards for sulfur oxides (sulfur dioxide) – Final decision,” May 22, 1996, p. 25568). Since 2010, there is only a 1-h standard in the U.S. for SO2 (Federal Register, “Primary National Ambient Air Quality Standard for sulfur dioxide,” June 22, 2010). For computational reasons, daily monitor readings are not first S. Luechinger / Journal of Health Economics 37 (2014) 219–231 227 Table 3 Robustness tests: Subsamples. Dependent variable Infant mortality rate Fixed-effects estimates SO2 R2 IV estimates SO2 2 R F-stat excl. instrument Control variables Year effects County effects Economic variables Climate variables No. of observations No. of counties No. of clusters Total (1) West (2) East (3) 1985–1990 (4) 1991–2003 (5) 0.026** (0.004) 0.27 0.019* (0.007) 0.31 −0.003 (0.009) 0.21 0.023* (0.010) 0.09 0.021** (0.005) 0.12 0.045** (0.009) 0.27 106.39 0.040** (0.012) 0.31 133.81 0.101 (0.115) 0.09 0.60 0.042* (0.017) 0.09 75.13 0.047** (0.014) 0.11 16.21 Y Y Y Y 7588 439 92 Y Y Y Y 6148 326 71 Y Y Y Y 1440 113 21 Y Y Y Y 1956 326 71 Y Y Y Y 5632 439 92 Notes: (1) OLS fixed-effects and IV fixed-effects regressions. The instrumental variable is the estimated reduction in SO2 pollution due to desulfurization at power plants for each county-year. It is estimated by summing over all power plants the product of a dummy variable with value one if a power plant operates at time t and zero otherwise, estimated pre-desulfurization emissions, a dummy variable with value one if a power plant has installed a scrubber at time t and zero otherwise, a distance decay function, and the frequency a county lies downwind of a power plant. The instrumental variable is included together with a control variable capturing the estimated effect of changes in the power plant population on SO2 pollution. It is estimated in a similar way as the instrumental variable but without the dummy variable for the installation of scrubbers. (2) The unit of observation is the county-year. (3) Cluster robust standard errors in parentheses allow for clustering at the level of 92 analytical regions encompassing several counties. (4) * is significant at the 95 percent level and ** at the 99 percent level. fixed-effects estimate based on the number of days the former U.S. 24-h standard is exceeded, I find for all these alternative measures statistically significant positive effects on infant mortality. However, the estimated effects are smaller with elasticities of 0.04 (fixed-effects estimate) and 0.09 (instrumental variable estimate) for the annual maximum concentration, 0.01 and 0.03 for the number of days the German 24-h standard is exceeded, and 0.002 and 0.02 for the number of days the former U.S. 24-h standard is exceeded.13 To further test the robustness of the results, I estimate the baseline regressions for several subsamples, namely for West Germany, East Germany, the 1985–1990 period, and the 1991–2003 period,14 and I control for additional confounding variables. Table 3 presents the results for the different subsamples. The estimates are comparable across subsamples with the exception of the East German subsample. In the East German subsample, SO2 pollution is unrelated to infant mortality. In case of the instrumental variable estimate, actual differences to other subsamples are more pronounced than it appears from the point estimate, since both the reduced form and the first stage regressions have the wrong sign and are imprecisely estimated. Therefore, neither measurements nor predicted changes of SO2 concentration have the expected effect on infant mortality in East Germany. To interpret this result, it is important to remember that the large improvements in air quality in East Germany coincided with large interpolated on a grid and then aggregated to the county level but rather directly interpolated to county centroids. 13 The means and estimated effects for the annual maximum concentration are 123.84 g/m3 (mean), 0.002 (std. err.: 0.001) (fixed-effects estimate), and 0.004 (std. err.: 0.001) (instrumental variable estimate). For the number of days in exceedance of the German standard the respective figures are 4.93 days, 0.014 (std. err.: 0.007), and 0.036 (std. err.: 0.009). For the number of days in exceedance of the former U.S. 24-h standard they are 0.36 days, 0.025 (std. err.: 0.035), and 0.255 (std. err.: 0.086). 14 Two reasons explain the uneven partition of the time periods. First, this partition roughly separates the main period of desulfurization in the West from the main period of desulfurization is the East. Second, the subsample for the period 1991–2003 corresponds to the sample of the robustness analysis based on microdata reported below. economic and socio-demographic changes in the aftermath of reunification. An alternative way to look at spatial heterogeneity is by estimating differential effects of SO2 on infant mortality with distance to the former East-West border. Fig. 5 presents the resulting estimates together with the baseline estimates reported in column 3 of Table 2. As can be seen, I find no evidence for differences in the fixed-effects estimates with distance to the inner German border. However, the instrumental variable estimate seems to be larger in the West compared to the East, although the 95 percent confidence interval encompasses the baseline estimate except for the western-most parts of Germany. Table 4 presents the results from regressions with additional control variables. For comparison, column 1 shows again the baseline estimates corresponding to column 3 of Table 2. In column 2, I control for the TSP concentration. SO2 and TSP concentrations are often correlated. Hence, without controlling for the TSP concentration, SO2 concentration may stand for air pollution at large. In Bobak and Leon (1999), the inclusion of TSP concentration renders the effect of SO2 concentration insignificant. This decrease in the effect of SO2 does not imply that SO2 pollution is irrelevant. SO2 is a precursor of TSP and, thus, the two pollutants are causally linked. However, as can be seen from Table 4, adding TSP concentration leaves the SO2 coefficient virtually unaffected. In column 3, I augment the models with year specific distance-to-city polynomials and year specific close-to-theEast-West-German-border effects. Year specific distance-to-city polynomials should control for urban/rural trends, year specific close-to-the-East-West-German-border effects for reunification related developments. I follow Redding and Sturm (2008) and define closeness as <75 km. They show that West German cities within this distance to the East-West German border suffered a sudden drop in population growth compared to other cities with the breakup of Germany after the Second World War but regained ground with the German reunification. Including these additional control variables affects the fixed-effects estimates little but slightly increases the instrumental variable estimate. In column 4, I add county specific linear time-trends. These time-trends flexibly account for changes in socio-demographic 228 S. Luechinger / Journal of Health Economics 37 (2014) 219–231 Table 4 Robustness tests: Additional controls. .04 R2 IV estimates SO2 .02 Fixed-effects estimates SO2 .03 Dependent variable Infant mortality rate 2 0 .01 Marginal effect of SO2 .05 A. Fixed-effects estimates -400 -200 0 200 Distance to border; negative for West Germany (2) (3) (4) 0.026** (0.004) 0.27 0.026** (0.004) 0.27 0.026** (0.005) 0.27 0.009 (0.008) 0.35 0.045** (0.009) 0.27 106.39 0.046** (0.010) 0.27 104.59 0.058** (0.010) 0.27 73.38 0.071** (0.027) 0.33 39.49 Y Y Y Y N N N N 7588 439 92 Y Y Y Y Y N N N 7588 439 92 Y Y Y Y N Y Y N 7588 439 92 Y Y Y Y N N N Y 7588 439 92 .05 .1 Notes: (1) OLS fixed-effects and IV fixed-effects regressions. The instrumental variable is the estimated reduction in SO2 pollution due to desulfurization at power plants for each county-year. It is estimated by summing over all power plants the product of a dummy variable with value one if a power plant operates at time t and zero otherwise, estimated pre-desulfurization emissions, a dummy variable with value one if a power plant has installed a scrubber at time t and zero otherwise, a distance decay function, and the frequency a county lies downwind of a power plant. The instrumental variable is included together with a control variable capturing the estimated effect of changes in the power plant population on SO2 pollution. It is estimated in a similar way as the instrumental variable but without the dummy variable for the installation of scrubbers. (2) The unit of observation is the countyyear. (3) Cluster robust standard errors in parentheses allow for clustering at the level of 92 analytical regions encompassing several counties. (4) ** is significant at the 99 percent level. 0 Marginal effect of SO2 .15 B. Instrumental variable estimate R F-stat excl. instrument Control variables Year effects County effects Economic variables Climate variables TSP Year spec. distance to city Year spec. close to E-W border County spec. time trends No. of observations No. of counties No. of clusters (1) -400 -200 0 200 Distance to border; negative for West Germany Fig. 5. Differential effects of SO2 on infant mortality with distance from former East–West border. Notes: The solid black lines depict differential effects of SO2 on infant mortality with distance from former East–West border, which are estimated in models with an interaction term of SO2 × distance from border but otherwise identical to the models in column 3 of Table 2. Red lines indicate the size of the baseline effects reported in column 3 of Table 2. Dashed lines denote the 95 percent confidence intervals for a standard normal distribution. (For interpretation of the references to color in this figure legend, the reader is referred to the web version of this article.) factors, environmental policies, and other factors affecting infant mortality. The inclusion of these time trends affects the fixed-effects and instrumental variable estimates in different directions: While the fixed-effects estimate is reduced by around 65 percent and becomes statistically insignificant, the instrumental variable estimate increases. There are two potential explanation for the decrease in the fixed-effects estimate caused by the inclusion of county specific time-trends. On the one hand, the decrease may indicate that the baseline results are biased by omitted variables. On the other hand, county specific time-trends may eliminate much of the identifying variation in the fixedeffects estimates and, thereby, render measurement errors more important. 4.4. Extensions As discussed by Chay and Greenstone (2003a,b), one mechanism through which air pollution affects infant mortality are adverse effects on fetal development. To assess the importance of this mechanism, they suggest looking at the effects of air pollution on neonatal mortality because deaths due to poor fetal development are likely to occur in the neonatal period. In this spirit, Table 5 presents the estimated effects of SO2 concentration on infant mortality within the first day (columns 1 and 4) and within the first 28 days (columns 2 and 5) after birth. Since I only have disaggregated county x year-level data for the period 1986–1999 and since microdata are only available for the period 1991–2003, Table 5 also reports the estimated effect for infant mortality within the first year for the respective periods (columns 3 and 6).15 The fixed-effects estimates suggest that the effect of air pollution on infant mortality is not larger in the neonatal period and that the effect even may be slightly smaller. There is no effect on deaths within the first day in either period and on deaths within the first 28 days in 1986–1999. Deaths within the first 28 days increase with an elasticity of 0.05 in 1991–2003, which corresponds to the elasticity of 0.05 for deaths within the first year found both in 1986–1999 and in 1991–2003. Similarly, the instrumental variable estimates too provide no evidence for a strongly disproportionate effect on mortality in the neonatal period. The respective elasticities are 0.05 in 1986–1999 and 0.09 in 1991–2003 for deaths within the first day, 0.10 in 1985–1999 and 0.11 in 1991–2003 for deaths within 15 The infant mortality data are inherently annual. Similarly, lacking more temporally disaggregated data on power plants, this is also true for the instrumental variable. Thus, all estimates are based on estimates of contemporaneous effects of air pollution on infant mortality and pregnancy outcomes. Using the contemporaneous level is also appropriate for assessing the effects of poor fetal development due to exposure during pregnancy since air pollution during the last trimester has been shown to be important for infant health (Currie et al., 2009). S. Luechinger / Journal of Health Economics 37 (2014) 219–231 229 Table 5 Infant mortality within 1 day, 28 days and 1 year and male and female infant mortality. Dependent variable Infant mortality rate Fixed-effects estimates SO2 R2 IV estimates SO2 R2 Control variables Individual and maternal variables Year effects County effects Economic variables Weather variables No. of observations No. of counties No. of clusters A. County × year-level data, 1986–1999 B. County × year × sex × legitimacy-level data, 1991–2003 W/in 1 day (1) W/in 28 days (2) W/in 1 year (3) W/in 1 day (4) W/in 28 days (5) W/in 1 year (6) Males (7) Females (8) −0.001 (0.002) 0.04 0.001 (0.004) 0.09 0.018** (0.006) 0.22 0.002 (0.002) 0.05 0.014** (0.004) 0.07 0.020** (0.005) 0.12 0.024** (0.006) 0.13 0.016** (0.005) 0.10 0.004 (0.004) 0.04 0.018* (0.008) 0.08 0.040** (0.011) 0.22 0.011 (0.008) 0.05 0.027* (0.012) 0.07 0.040* (0.017) 0.12 0.042* (0.019) 0.13 0.040* (0.019) 0.10 – Y Y Y Y 5481 439 92 – Y Y Y Y 5481 439 92 – Y Y Y Y 5481 439 92 Y Y Y Y Y 22,500 439 92 Y Y Y Y Y 22,500 439 92 Y Y Y Y Y 22,500 439 92 Y Y Y Y Y 11,250 439 92 Y Y Y Y Y 11,250 439 92 Notes: (1) OLS fixed-effects and IV fixed-effects regressions. The instrumental variable is the estimated reduction in SO2 pollution due to desulfurization at power plants for each county-year. It is estimated by summing over all power plants the product of a dummy variable with value one if a power plant operates at time t and zero otherwise, estimated pre-desulfurization emissions, a dummy variable with value one if a power plant has installed a scrubber at time t and zero otherwise, a distance decay function, and the frequency a county lies downwind of a power plant. The instrumental variable is included together with a control variable capturing the estimated effect of changes in the power plant population on SO2 pollution. It is estimated in a similar way as the instrumental variable but without the dummy variable for the installation of scrubbers. (2) The unit of observation is the county-year in Panel A and the county × year × sex × legitimacy-cell in Panel B. Regressions in Panel B are weighted by the number of live births in each cell. (3) Cluster robust standard errors in parentheses allow for clustering at the level of 92 analytical regions encompassing several counties. (4) * is significant at the 95 percent level and ** at the 99 percent level. Source of mortality data in Panel B: Research Data Centres of the Federal Statistical Office and the statistical offices of the Länder, statistics of births and statistics of deaths, 1991–2003; own analysis. See text for other sources. the first month, and 0.12 in 1985–1999 and 0.09 in 1991–2003 for deaths within the first year. Overall, these estimates do not suggest that poor fetal development caused by exposure during gestation is an important biological mechanism. As in other studies (Jayachandran, 2009; Tanaka, 2010), gender differences are relatively small. For males the estimated elasticities amount to 0.05 (fixed-effects estimate) and 0.09 (instrumental variable estimate), for females to 0.04 (fixed-effects estimate) and 0.10 (instrumental variable estimate). In order to further explore potential effects of exposure to air pollution during pregnancy, Table 6 presents estimates on the effect of SO2 on the number of stillbirths (per 1000 births) (column Table 6 Birth outcomes. Dependent variable Fixed-effects estimates SO2 R2 IV estimates SO2 R2 Control variables Individual and maternal variables Year effects County effects Economic variables Weather variables No. of observations No. of counties No. of clusters Stillbirths (1) Weight (2) SGA, weight (3) Length (4) SGA, length (5) 0.005( *) (0.003) 0.08 −0.104 (0.143) 0.86 0.024 (0.016) 0.23 0.001 (0.001) 0.89 0.061* (0.026) 0.33 −0.006 (0.011) 0.08 −0.540 (0.415) 0.86 0.093( *) (0.051) 0.22 −0.001 (0.002) 0.89 0.195** (0.060) 0.33 Y Y Y Y Y 22,500 439 92 Y Y Y Y Y 22,500 439 92 Y Y Y Y Y 22,500 439 92 Y Y Y Y Y 22,500 439 92 Y Y Y Y Y 22,500 439 92 Source of birth variables: Research Data Centres of the Federal Statistical Office and the statistical offices of the Länder, statistics of births and statistics of deaths 1991–2003; own analysis. See text for other sources. Notes: (1) OLS fixed-effects and IV fixed-effects regressions. The instrumental variable is the estimated reduction in SO2 pollution due to desulfurization at power plants for each county-year. It is estimated by summing over all power plants the product of a dummy variable with value one if a power plant operates at time t and zero otherwise, estimated pre-desulfurization emissions, a dummy variable with value one if a power plant has installed a scrubber at time t and zero otherwise, a distance decay function, and the frequency a county lies downwind of a power plant. The instrumental variable is included together with a control variable capturing the estimated effect of changes in the power plant population on SO2 pollution. It is estimated in a similar way as the instrumental variable but without the dummy variable for the installation of scrubbers. (2) SGA stands for small for gestational age. The variables here are similar in spirit to this measure but gestational age is unknown. Instead, the variables are defined here as birth weight or length more than two standard deviations below the mean of the birth cohort. (3) The unit of observation is the county × year × sex × legitimacy-cell. Regressions are weighted by the number of births in each cell in column 1 and by the number of live births in each cell in columns 2–5. (4) Cluster robust standard errors in parentheses allow for clustering at the level of 92 analytical regions encompassing several counties. (5) ( *) is significant at the 90 percent level, * at the 95 percent level, and ** at the 99 percent level. 230 S. Luechinger / Journal of Health Economics 37 (2014) 219–231 1), average birth weight (column 2), the number of infants with a birth weight of more than two standard deviations below the mean of their birth cohort (per 1000 live births) (column 3), average length at birth (column 4), and the number of infants whose length is more than two standard deviations below the mean of their birth cohort (per 1000 live births) (column 5). According to the fixed-effects estimate the frequency of stillbirths increases with SO2 pollution, but this finding does not carry over to the instrumental variable estimate. Conversely, the instrumental variable estimate but not the fixed-effects estimate suggests that SO2 pollution increases probability of infants with low weight, though the effect is imprecisely estimated. The strongest effects are found for the chance that infants have comparatively short length at birth. Interestingly, this finding is consistent with a study on air pollution and infant health for Germany by Coneus and Spiess (2012). Combining survey data and high-frequency pollution data for 2002–2007, they find that SO2 pollution negatively affects length at birth but not birth weight. In sum, the evidence suggests that exposure to SO2 during gestation affects fetal development and infant health at birth, but that poor fetal development is not the main mechanisms through which SO2 affects infant mortality. 5. Conclusion The results of this paper suggest that the desulfurization at power plants entailed considerable benefits in terms of infant health. Thus, they are directly relevant for the retrospective evaluation of this policy in Germany and, arguably, policies in other developed countries such as the emission trading program in the U.S. They also point to potentially large gains from cleaning up the air in developing countries with often very high levels of air pollution. This is all the more true as improved infant health is only one benefit of air quality among others. The estimated elasticities between SO2 pollution and infant mortality are at the lower end of published elasticities between pollution and infant mortality. Still, the very large reductions in SO2 pollution in developed countries in the last century imply large cumulative effects. In future research it would be interesting to disentangle the various reasons behind differences in estimated effects of pollution. Potential reasons range from differences in the toxicity of pollutants, differences in how ambient air pollution translates to individual exposure, to contextual factors that differ across countries and influence individuals’ exposure and susceptibility to air pollution. References Ashenfelter, O., Greenstone, M., 2004. Using mandated speed limits to measure the value of a statistical life. Journal of Political Economy 112 (S1), S226–S267. Ashmore, M.R., Dimitroulopoulou, S., 2009. Personal exposure of children to air pollution. Atmospheric Environment 43 (1), 128–141. Bakkum, A., Bartelds, H., Duiser, J.A., Veldt, C., 1987. Handbook of Emission Factors, Part 3. Stationary Combustion Sources. Ministry of Housing, Physical Planning and Environment, The Hague, NL. Bobak, M., Leon, D.A., 1992. Air pollution and infant mortality in the Czech Republic, 1986–88. Lancet 340 (8826), 1010–1014. Bobak, M., Leon, D.A., 1999. The effect of air pollution on infant mortality appears specific for respiratory causes in the postneonatal period. Epidemiology 10 (6), 666–670. Bresnahan, B.W., Dickie, M., Gerking, S., 1997. Averting behavior and urban air pollution. Land Economics 73 (3), 340–357. Chay, K.Y., Greenstone, M., NBER Working Papers No. 10053 2003a. Air quality, infant mortality, and the Clean Air Act of 1970. National Bureau of Economic Research, Cambridge, MA. Chay, K.Y., Greenstone, M., 2003b. The impact of air pollution on infant mortality: evidence from geographic variation in pollution shocks induced by a recession. Quarterly Journal of Economics 118 (3), 1121–1167. Chinn, S., du, C., Florey, V., Baldwin, I.G., Gorgol, M., 1981. The relation of mortality in England and Wales 1969–73 to measurements of air pollution. Journal of Epidemiology and Community Health 35 (3), 174–179. Coneus, K.C., Spiess, K., 2012. Pollution exposure and child health: evidence for infants and toddlers in Germany. Journal of Health Economics 31 (1), 180–196. Crocker, T.D., Schulze, W., Ben-David, S., Kneese, A.V., 1979. Methods development for assessing air pollution control benefits. Experiments in the Economics of Air Pollution Epidemiology, vol. I. Environmental Protection Agency, Washington. Currie, J., Gruber, J., 1996. Saving babies: the efficacy and cost of recent changes in the Medicaid eligibility of pregnant women. Journal of Political Economy 104 (6), 1263–1296. Currie, J., Neidell, M., 2005. Air pollution and infant health: what can we learn from California’s recent experience? Quarterly Journal of Economics 120 (3), 1003–1030. Currie, J., Neidell, M., Schmieder, J.F., 2009. Air pollution and infant health: lessons from New Jersey. Journal of Health Economics 28 (3), 688–703. Currie, J., Schmieder, J.F., 2009. Fetal exposures to toxic releases and infant health. American Economic Review. Papers and Proceedings 99 (2), 177–183. Dales, R., Burnett, R.T., Smith-Doiron, M., Stieb, D.M., Brook, J.R., 2004. Air pollution and sudden infant death syndrome. Pediatrics 113 (6), e628–e631. Deschênes, O., Greenstone, M., 2011. Climate change, mortality, and adaptation: evidence from annual fluctuations in weather in the US. American Economic Journal: Applied Economics 3 (4), 152–185. Deschênes, O., Greenstone, M., Shapiro, J.S., NBER Working Paper No. 18267 2012. Defensive Investments and the Demand for Air Quality: Evidence from the NOx budget program and ozone reductions. National Bureau of Economic Research, Cambridge, MA. Foster, A., Gutierrez, E., Kumar, N., 2009. Voluntary compliance, pollution levels, and infant mortality in Mexico. American Economic Review. Papers and Proceedings 99 (2), 191–197. Graff Zivin, J., Neidell, M., 2009. Days of haze: environmental information disclosure and intertemporal avoidance behavior. Journal of Environmental Economics and Management 58 (2), 119–128. Graff Zivin, J.S., Neidell, M., 2013. Environment, health, and human capital. Journal of Economic Literature 51 (3), 689–730. Gruber, J., 1997. Health insurance for poor women and children in the U.S: lessons from the past decade. In: James Poterba (Ed.), Tax Policy and the Economy, vol. 11. MIT Press, Cambridge, MA, pp. 169–211. Ha, E.-H., Lee, J.-T., Kim, H., Hong, Y.-C., Lee, B.-E., Park, H.-S., Christiani, D.C., 2003. Infant susceptibility of mortality to air pollution in Seoul, South Korea. Pediatrics 111 (2), 284–290. Hajat, S., Armstrong, B., Wilkinson, P., Busby, A., Dolk, H., 2007. Outdoor air pollution and infant mortality: analysis of daily time-series data in 10 English cities. Journal of Epidemiology and Community Health 61 (8), 719–722. Hedley, A.J., Wong, C.-M., Tach, T.Q., Ma, S., Tai-Hing Lam, H., Anderson, R., 2002. Cardiorespiratory and all-cause mortality after restrictions on sulphur content of fuel in Hong Kong: an intervention study. Lancet 360 (9346), 1646–1652. Hickey, R.J., Boyce, D.E., Clelland, R.C., Bowers, E.J., Slater, P.B., Technical Report No. 15 1976. Demographic and chemical variables related to chronic disease mortality in man. Department of Statistics, The Wharton School, University of Pennsylvania, Philadelphia. Jayachandran, S., 2009. Air quality and early-life mortality: evidence from Indonesia’s wildfires. Journal of Human Resources 44 (4), 916–954. Joyce, T.J., Grossman, M., Goldman, F., 1989. An assessment of the benefits of air pollution control: the case of infant health. Journal of Urban Economics 25 (1), 32–51. Lacasaña, M., Esplugues, A., Ballester, F., 2005. Exposure to ambient air pollution and prenatal and early childhood health effects. European Journal of Epidemiology 20 (2), 183–199. Lin, C.A., Pereira, L.A.A., Nishioka, D.C., Conceição, G.M.S., Braga, A.L.F., Saldiva, P.H.N., 2004. Air pollution and neonatal deaths in São Paulo, Brazil. Brazilian Journal of Medical and Biological Research 37 (5), 765–770. Lipfert, F.W., Zhang, J., Wyzga, R.E., 2000. Infant mortality and air pollution: a comprehensive analysis of U.S. data for 1990. Journal of the Air & Waste Management Association 50 (8), 1350–1366. Lleras-Muney, A., 2010. The needs of the Army. Using compulsory relocation in the military to estimate the effect of air pollutants on children’s health. Journal of Human Resources 45 (3), 549–590. Luechinger, S., 2009. Valuing air quality using the life satisfaction approach. Economic Journal 119 (536), 482–515. Mendelsohn, R., Orcutt, G., 1979. An empirical analysis of air pollution dose-response curves. Journal of Environmental Economics and Management 6 (2), 85–106. Mittelbach, G., 1991. Desulfurization of flue gases on the basis of lime or limestone scrubbing. In: Daniel van Velzen (Ed.), Sulphur Dioxide and Nitrogen Oxides in Industrial Waste Gases: Emission, Legislation and Abatement. Springer, Dordrecht, pp. 93–109. Moretti, E., Neidell, M., 2011. Pollution, health, and avoidance behavior: evidence from the ports of Los Angeles. Journal of Human Resources 46 (1), 154–175. Neidell, M., 2009. Information, avoidance behavior, and health: the effect of ozone on asthma hospitalizations. Journal of Human Resources 44 (2), 450–478. Pope, C.A., Burnett, R.T., Thun, M.J., Calle, E.E., Krewski, D., Ito, K., Thurston, G.D., 2002. Lung cancer, cardiopulmonary mortality, and long-term exposure to fine particulate air pollution. Journal of the American Medical Association 287 (9), 1132–1141. S. Luechinger / Journal of Health Economics 37 (2014) 219–231 Redding, S.J., Sturm, D.M., 2008. The costs of remoteness: evidence from German division and reunification. American Economic Review 98 (5), 1766–1797. Schärer, B., Haug, N., 1990. Bilanz der Großfeuerungsanlagen-Verordnung. Staub. Reinhaltung der Luft 50, 139–144. Schulz, W., 1985. Der monetäre Wert besserer Luft: Eine empirische Analyse individueller Zahlungsbereitschaft und ihrer Determinanten auf der Basis von Repräsentativumfragen. Lang, Frankfurt a.M., Bern/New York. Schwartz, J., Marcus, A., 1990. Mortality and air pollution in London: a time series analysis. American Journal of Epidemiology 131 (1), 185–194. Schwartz, Stephen E., 1989. Acid deposition: unraveling a regional phenomenon. Science 243 (4892), 753–763. Shinkura, R., Fujiyama, C., Akiba, S., 1999. Relationship between ambient sulfur dioxide levels and neonatal mortality near the Mt. Sakurajima volcano in Japan. Journal of Epidemiology 9 (5), 344–349. Šram, R.J., Binkova, B., Dejmek, J., Bobak, M., 2005. Ambient air pollution and pregnancy outcomes: a review of the literature. Environmental Health Perspectives 113 (4), 375–382. 231 Summers, P.W., Fricke, W., 1989. Atmospheric decay distances and times for sulphur and nitrogen oxides estimated from air and precipitation monitoring in Eastern Canada. Tellus 41B (3), 286–295. Tanaka, S., 2010. Air Pollution, Infant Mortality, and the Environmental Regulations in China. Boston University, Mimeo. Traup, S., Kruse, B., 1996. Wind und Windenergiepotentiale in Deutschland. Winddaten für Windenergienutzer. Deutscher Wetterdienst, Offenbach am Main, DE. UBA, 2012. National trend tables for the German atmospheric emission reporting, 1990–2011. http://www.umweltbundesamt.de/themen/luft/emissionenvon-luftschadstoffen Viscusi, W.K., Aldy, J.E., 2003. The value of a statistical life: a critical review of market estimates throughout the world. Journal of Risk and Uncertainty 27 (1), 5–76. Woodruff, T.J., Darrow, L.A., Parker, J.D., 2008. Air pollution and postneonatal infant mortality in the United States, 1999–2002. Environmental Health Perspectives 116 (1), 110–115.
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