John Guido, PT, DPT, ATC, SCS, CSCS Ochsner Health System INTRODUCTION Very common, very frustrating problem in the thrower’s shoulder Shoulder already functioning at the extremes of motion Wide spectrum of pathology that can cause instability, pain and dysfunction INTRODUCTION Injuries to the biceps – labrum complex SLAP Lesions - Superior labral anterior to posterior tears are recognized as a major factor in the dysfunction of the disabled throwing shoulder Systematic lit review Kibler and Sciascia Arthroscopy 2016 No consensus regarding current practice in the Rx of SLAP lesions Wide variability Treatment approaches Surgical indications Surgical techniques Rehabilitation programs BACKGROUND Epidemiology Difficult to determine exact numbers but in one sports med practice, SLAP repairs comprised 6.3% of all s’ surgeries Modifications to Snyder’s original classification are such that there are now 10 different types of SLAP lesion variations (Modaressi S et al AJR 2011) Overall, SLAP repair numbers may be decreasing Overall, age of patient is decreasing “If I see it, I fix it” ANATOMY Average depth of the glenoid is doubled by presence of labrum Chock block stabilizing effect to limit translation Labrum increases the total surface area for articulation enhancing concavity-compression effects. Aids in the attachment of the glenohumeral ligaments to the glenoid. The superior labrum is looser and more mobile than its inferior counterpart WHAT EXACTLY IS A SLAP LESION? A tear in the superior labrum region. Between 10 & 2 on a clock face. Related to pull on the origin site of the long head of the biceps. First described by Andrews in 1985 in overhead athletes fraying, detachment, tearing of biceps SLAP Lesions-Classification I II III IV Fraying & Degeneration Detachment of labrum and biceps -UNSTABLE Bucket handle tear w/ rim of intact sup. labrum Bucket handle tear extending into biceps but anchor intact BIOMECHANICS Provides 10% overall stability in the shoulder Labrum acts as a biofeedback mechanism to RC, will tell it when to contract if shoulder goes too far Increased GHJ translation and loading of the long head of the biceps tendon after SLAP lesions (Patzer et al 2012) The tensile strength of the labrum is less than the capsule, so it is more prone to injury with anterior stress. SLAP LESION Causes significant loss in static stability but how?? (therefore, dynamic stability will ultimately compromised) 100 - 120% increase in strain on the AIGHL 11 – 19% decrease in the GHJ’s ability to withstand rotational force Therefore… significant increase in the load on the capsular ligaments (Cheng & Karzel 1997) MECHANISM OF INJURY IN THROWERS / OH ATHLETES Andrews 1985 Burkhart, Morgan, Kibler Jobe Several theories exist to explain the MOI of SLAP tears but the exact cause has not been identified Most likely multifactorial May exist on a spectrum of injuries in a thrower’s shoulder since an isolated SLAP lesion is usually not seen SLAP lesions lead to GHJ Instability (Support of the superior GH ligament is lost) MECHANISM OF INJURY IN THROWERS / OH ATHLETES So, let’s take a look at SLAP lesions as they could occur in the thrower’s shoulder based on the phase of throwing In other words, marry the theory to the practice WIND UP/EARLY COCKING/STRIDE WEED PULLING = HORIZ ABDUCTION LATE COCKING/MAX ER Burkhart, Morgan, Kibler “peel-back” mechanism as the cause of SLAP lesion. When the shoulder is placed in a position of abduction and maximum ER, the rotation produces a twist at the base of the biceps – torsional force applied to the anchor. Peak strain occurs in this position. GIRD/Scapular dyskinesis LATE COCKING / MAX ER / ACCELERATION Jobe and Jobe Internal impingement / anterior instability Over application of normal glenohumeral contact either on an acute or a chronic basis Increased frequency or increased load leads to damage to 1 or more of 5 tissues: (1) superior labrum (2) biceps origin—internal fibers of rotator cuff (PASTA) (3) bony glenoid (4)greater tuberosity (5) inferior glenohumeral ligament. LATE COCKING / MAX ER / ACCELERATION Jobe and Jobe More than 1 of these tissues is damaged producing a variable pattern of injury Continued loading after the development of pain will lead to stretching of the inferior glenohumeral ligament and subsequent instability and the need for surgical reconstruction DECELERATION / FOLLOW THROUGH Andrews 1985 AJSM The extremely high velocity of elbow extension which is generated must be decelerated through the final 30 degrees of elbow extension Of the muscles of the arm that provide the large deceleration forces in the follow-through phase of throwing, only the biceps brachii traverses both the elbow joint and the shoulder joint Additional forces are generated in the biceps tendon in its function as a "shunt" muscle to stabilize the glenohumeral joint during the throwing act SYMPTOMS Cluster of signs and symptoms Loss of IR Pain with OH motions Loss of RC/scap strength and endurance Mechanical symptoms Loss of velocity/control/”dead arm” Inability to “loosen up” (Manske 2010) *”Where in the throwing motion do you have pain?” Cocking Acceleration After ball release Labral Examination – Special Tests based on MOI “Weed pulling” Clunk test The examiner proximal hand provides an anterior translation of the humeral head while simultaneously rotating the humerus externally with the hand holding the elbow. Attempt to trap the labrum. A positive test is produced by the presence of a clunk or grinding sound Labral Examination – Special Tests based on MOI Peel-back injury (overhead athlete) Biceps Load Biceps Load Test The shoulder is placed in 90º of abduction and maximal ER. At maximal ER and with the forearm in a supinated position, the patient is instructed to perform a biceps contraction against resistance. A positive finding for a SLAP lesion is deep pain within the shoulder. Labral Examination-Special Tests Pronated Load Test Same as Biceps Load test however, the forearm is in the fully pronated position to increase the tension on the biceps and subsequently the labral attachment. When maximum ER is achieved, the patient is instructed to perform an isometric contraction of the biceps. Simulates the “peel-back” mechanism. Labral Examination – Special Tests based on MOI Jobe anterior instability / Int Impingement Relocation test (+ relieves posterior pain) Anterior drawer Labral Examination – Special Tests based on MOI Follow through eccentric load Active Compression and SLAPrehension tests SLAPrehension test Berg and Cuillo O’Brien test but at 45 degrees Hadd The increased adduction is thought to increase stress on the biceps tendon but have to watch AC joint IMAGING - MRA SLAP Lesions- Treatment Type I: debridement Type II: reattachment of unstable biceps anchor to the glenoid rim using suture anchor Type III: excision vs repair of bucket handle tear of the labrum Type IV: excision of torn labrum and biceps tear vs suturing the split if >50% biceps torn: biceps tenodesis Arthroscopic Repair of Type II Slap Lesion WHY FIX A SLAP LESION? SLAP repair without associated LHB tenotomy helps normalize GH translation (Patzer et al JSES 2012) Rodosky et al the LHB contributes to anterior GH stability by increasing the shoulder’s resistance to torsional forces in the abd/ER position, diminishing stress on the IGHL What we see clinical when we do PROM into abd/ER at 90-90 the bicep fires to guard the shoulder (this is decreased if scapula is correctly positioned) SOMETHING TO CONSIDER…. McCulloch et al Arthroscopy 2013 Cadaveric study 7 shoulders with SLAP repair Anterior anchor had the greatest effect on ER The presence of 1 or 2 anchors posterior to the biceps did not have a significant effect on rotation When performing SLAP repairs on baseball pitchers, surgeons should consider avoidance of the anchor anterior to the biceps OVERVIEW OF REHABILITATION Removal from offending activity Stretch posterior capsule/cuff Rotator cuff/scapular strengthening/endurance program Shoulder stabilization program UE Prime mover/core/LE strengthening UE plyometrics/prethrowing program Interval throwing program Video biomechanical analysis Fault correction Long toss and mound program Interval hitting program Rehabilitation: Reattachment POD 1 SLING x 4 weeks ELBOW, WRIST , HAND AROM SCAPULAR REPOSITIONING PROM/ROM check: 90/90 POS/45/50 (ER/IR @ 45 abd) ER 10 degrees to 45 degrees by week 4 IR to tolerance up to 50 degrees by week 4 Rehabilitation: Reattachment Pendulum swings S’ isometrics at 2 weeks except Flexion Rhythmic stabilization at week 2 45 deg abd in ER/IR No stress on biceps and no closed chain positions Stationary bike/LE/core training Rehabilitation: Reattachment POW 5-6 Begin to work on restoring FULL THROWER”S ROM Focus on stretching posterior capsule CONTINUE STRENGTHENING PROGRAM Rotator cuff / scapular program CONTINUE SHOULDER STABILIZATION PROGRAM No isolated bicep strengthening x 8 weeks Rehabilitation: Reattachment Assess for possible GIRD – glenohumeral internal rotation deficit Loss of > 30-40 degrees of IR Burkhart et al GIRD found in 100% of a series of 124 symptomatic baseball throwers found to have a SLAP tear arthroscopically (Non op Rx GIRD 2 weeks) Rehabilitation: Reattachment Decreased posterior capsular mobility (post band of IGHL) causes a posterior superior shear during elevation and ER This is due to obligate translation as described by Harryman in 1990 and Karduna in 1996 An obligate translation is one that occurs following a tightness in capsular tissues that causes motion of a given joint segment to occur in the direction opposite the tight joint structures Rehabilitation: Reattachment Thus, an isolated tight IGHL complex will create an obligate translation of the humeral head in a superior direction with elevation and a tight posterior capsule will create an anterior shear with IR POSTERIOR CAPSULE STRETCH SLEEPER HANG 2-3 lbs 1’x2 Rehabilitation: Reattachment FULL THROWER”S ROM by 10-12 weeks Weeks 12-16 UE plyometrics and pre throwing program Week 16 -24 return to throwing program Weeks 24-28 mound program Week 28 Release to full activity CORE TRAINING: ROLLING SERIES Arm patterns Leg Patterns LOWER EXTREMITY TRAINING OVERHEAD DEEP SQUAT UE PLYOMETRICS Chops 3x10 R/L Lifts 3x10 R/L OUTCOMES Weber et al – only 26% of pts stated that they were pain-free and only 13% rated their function as normal Boileau et al only 20% pts were able to return to sports at their preinjury level Gorantla et al - % of good to excellent results after SLAP repair ranged from 40-94% Return to play ranged from 20-94% and in OH athletes 22-64% for baseball Ide et al Baseball 12 of 19 cases (63%) were able to return to their preinjury level (36% failure rate) OUTCOMES Neri et al AJSM 2009 - Time to return to sport 8,45 months Brockmeyer et al – results of SLAP repair in the throwing athlete is far less successful with a significant number of pts who will not regain their preinjury level of performance OUTCOMES Chalmers et al - 18 pitchers (7 uninjured controls, 6 SLAP repairs, 5 subpectoral tenodesis (KSSTA 2016) Altered T’ rotation in the SLAP repair subjects Concluded that even in high level athletes, biceps tenodesis is a reliable option Boileau et al AJSM 2009 – biceps tenodesis for a type II SLAP lesion showed a 93% satisfaction rate and an 87% return to their previous level of sport compared with only 20% after SLAP repair OUTCOMES Mollon B et al Arthroscopy 2016 10% of pts who underwent isolated SLAP repair required at 2nd procedure SAD 35% Debridement 27% Repeat SLAP repair 20% Biceps tenodesis or tenotomy 13% Patients < 20 yo after isolated SLAP repair more likely to undergo Bankart repair males > females (80% of cases) CONCLUSIONS SLAP lesions occur on a spectrum of pathology in the thrower’s shoulder Not a guarantee that an athlete will be able to return to their 100% preinjury level of function Restore full thrower’s motion Correct scapular dyskinesis Create dynamic stability Don’t forget about the LE / core Identify and correct biomechanical throwing faults “If you see it, fix it”
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