Small sizes and indolent evolutionary dynamics challenge
the potential role of P2RY8-CRLF2–harboring clones as
main relapse-driving force in childhood ALL
by Maria Morak, Andishe Attarbaschi, Susanna Fischer, Christine Nassimbeni,
Reinhard Grausenburger, Stephan Bastelberger, Stefanie Krentz, Gunnar Cario,
David Kasper, Klaus Schmitt, Lisa J. Russell, Ulrike Pötschger, Martin Stanulla,
Conny Eckert, Georg Mann, Oskar A. Haas, and Renate Panzer-Grümayer
Blood
Volume 120(26):5134-5142
December 20, 2012
©2012 by American Society of Hematology
Identification of P2RY8-CRLF2 in childhood BCP-ALL cases.
Maria Morak et al. Blood 2012;120:5134-5142
©2012 by American Society of Hematology
Quantification of P2RY8-CRLF2 genomic breakpoints and CRLF2 transcripts.
Maria Morak et al. Blood 2012;120:5134-5142
©2012 by American Society of Hematology
Clinical outcome of children with BCP-ALL enrolled in the BFM ALL 2000 protocol in Austria
according to P2RY8-CRLF2.
Maria Morak et al. Blood 2012;120:5134-5142
©2012 by American Society of Hematology
Patterns of genomic P2RY8-CRLF2 and CRLF2 transcripts of fusion-positive ALL relapse cases.
Maria Morak et al. Blood 2012;120:5134-5142
©2012 by American Society of Hematology
Detection of the P2RY8-CRLF2 in neonatal blood spots.
Maria Morak et al. Blood 2012;120:5134-5142
©2012 by American Society of Hematology