September 2013 - Clint Publications

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VOL. 20, NO. 3
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ISSN 15294722
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SEPTEMBER 2013
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Optimal Thyroid
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These statements have not been evaluated by the Food and Drug Administration. These products are not intended to diagnose, treat, cure, or prevent any disease.
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
……. …….
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
Spring 2006
SEPTEMBER
MARCH 2011
2013


 

402 West County Road D
Saint Paul, MN 55112
877-474-5767
Jennifer A. Collins, Ph. D.
Karla Walker, Pharm. D.
Mark G. Catlin, M. D.

Patient Test Order Information
PINC-POWER MED INC
#16 MUNICIPAL DR STE E
ARNOLD, MO 63010
Name:
Patient ID:
Patient Phone:
DOB:
Sex:
MALONEY,JACQUELYN
08/16/1935
Age: 77
Collected:
Received:
Reported:
06/27/2013
06/29/2013
07/08/2013
1:47 PM
9:10 AM
Report Status:
FINAL




Account #: 50453817
Ordered By:
Requisition #: MA1099431
Accession #: F5416438
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
Test
Result
Flag
VAP CHOLESTEROL
TOTAL LDL-CHOL
220
H
(Desirable range <100 mg/dL for CHD, Diabetes, or its
equivalent)
TOTAL HDL-CHOL
69
TOTAL VLDL-CHOL
44
H
TOTAL CHOL
333
H
TRIGLYCERIDES
245
H
TOTAL APO B100
164
H
NON-HDL CHOL
264
H
Lp(a) CHOL
9.0
IDL CHOL
49
H
LDL-R (Real)-C
162
H
TOTAL LDL-C
220
H
REAL-LDL SIZE PATTERN
A
[__________________]
Pattern B
Small, Dense LDL
[_______]
Pattern
A/B
Units
Reference Range
mg/dL
<130
mg/dL
mg/dL
mg/dL
mg/dL
mg/dL
mg/dL
mg/dL
mg/dL
mg/dL
mg/dL
>=40
<30
<200
<150
<109
<160
<10
<20
<100
<130
A
[______*________]
Pattern A
Large Buoyant LDL
REMNANT LIPOPROTEINS
72
H
mg/dL
Due to the presence of additional risk factors, consider lowering
LDL-C goal
CONSIDERATION
NO
HDL-2
26
mg/dL
HDL-3
44
mg/dl
VLDL-3
23
H
mg/dL
LDL4
0.0
mg/dL
LDL3
38.5
mg/dL
LDL2
57.7
mg/dL
LDL1
66.2
mg/dL
<30
No
GENDER NEEDED
GENDER NEEDED
<10
Analysis performed by Atherotech Inc; 201 London Parkway
Suite 400, Birmingham, AL 35211




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vC2.2
Certified Integrative
Chiropractic Physician
g
n
i
m
o
C
n
o
So
100 Hour Certification Program
The philosophy of integrative medicine
Integrative assessment of patient
history and physical exam
The integrative approach to disease
Disease prevention
Clinical botanical medicine
Clinical homeopathy
Clinical nutrition
Conservative care for adult and
pediatric acute and chronic conditions
Basic and advanced blood chemistries
Advanced functional diagnostic testing
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□ Charlotte, NC □ Baltimore. MD □ Dallas. TX □ Omaha, NE [email protected]
□ Pittsburgh, PA □ Springfield, NJ □ Canton, OH □ Chicago. IL www.drkessinger.com
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  Cervical nerve root
irritation, from any reason, may not only cause
headaches but also a chronic, generalized hypertonicity
with accompanying somatic cervicobrachial
symptomatology.



  Cluster headaches (CH) are
characterized by sudden paroxysmal attacks of severe

head pain that occur in a collection of individual
assaults experienced over days, weeks, or even months.

An abrupt and total remission from attacks may last for
a year or more with recurrences possible at anytime

without warning. CH is far more common in men and
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

         
       

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       
         
       
            

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           
         
          



         

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
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

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Headache is one of the most common disorders, and by
far, the most common neurological complaint. In all
chronic headache patients, suspect spinal subluxations
and allergies. Headaches are divided into two groups;
functional and pathological. Functional headaches are
usually chronic and not generally regarded as
dangerous. Pathological headaches are usually caused
by an underlying condition, such as trauma.
  The most frequent include
tension headache, vertebrogenic headache, cluster
headache, sinus headache, vascular headache, and
temporal arteritis.
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  A nonspecific headache
characterized by a dull pain reported throughout the
head. The patient will usually report neck pain with
this type of headache.
SEPTEMBER 2013
have a selective sensitivity to a variety of substances
that precipitate or aggravate the headache. Tobacco
smoke, alcohol, stress, perfumes, oversleeping, fatigue,
environmental exposure and neck flexion and rotation.
CH frequently coincide with the seasons, indicating
allergies. Other conditions reported to be associated
with CH include gastrointestinal symptoms, transient
muscular twitching, paresthesia, and arrhythmias. CH
victims are usually restless, preferring to walk around
during the attack.
 The cause of CH is speculated to be either
neurological and/or vascular. Clearly both are involved.
One hypothesis postulates the pathology affects the
brainstem causing a parasympathetic stimulation of the
vagus nerve giving rise to the various symptoms.
  The sinus headache may be
associated with infection, inflammation, and/or
congestion of the nasal sinuses of the head. The pain is
generally described as dull or gnawing that may range
from mild to severe. The pain is characteristically
localized over the affected part. Sinus headaches may
be misdiagnosed as CH's. In CH the unilateral pain
appears suddenly, is severe and constant, and
drastically increased by reclining.
  The vascular headache is
characterized by a throbbing pain which is often
synchronized with the pulse, and can be serious and
disabling. Most vascular headaches occur due to the
dilation of the extracranial arteries that are branches of
the carotid arteries and located mainly in the scalp.
The vascular headache is divided into two classes, 
 and .
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  Many different clinical types of
migraine are described in the literature. All migraine
headaches are characterized by severe, throbbing pain
that is customarily unilateral. They are often associated
with anorexia, nausea, vomiting, dizziness, cold hands,

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Dr. Jay Kessinger
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
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
As Governor Mike Huckabee stated on his syndicated
show, “

Unhealthiness is becoming the norm’ and not the
exception. Rumors abound of how our children will
not experience the longevity that we’ll have, yet at the
same time, through our increased longevity, we are
more apt than our parents to be plagued with
disabilities. Many of these haven’t been known to
humanity before. The toxic world in which we reside
(laden with so many nonnutritious food sources, less
physically exerting task masters, and charlatan crazes)
is leading us down a potentially physiologically
destructive pathway.
Several theories presently abound (from a full spectrum
of think tanks) toward reaching a solution of our
predominantly manmade dilemma of untimely
sickness and demise. One such theory is to return to
the primitive conditions from the times of yore. The
problem with this is its inherent lack of creature
comforts that we’ve come so accustomed to that they
are considered a necessity rather than an extravagance.
Another idea is to reside within a bubble, continually
being protected from harmful toxins of the outside
world. An obvious fallacy of this model is twofold.
Firstly, there is the impossibility of absolute separation
from the bacterial, et.al. infested microscopic world of
nature at its tiniest. Most importantly, there cannot be a
strong constitution of health without stress; i.e.,
immunological, neurological, and physiological.
Medicinally, our health maladies are treated singularly
with little attention being paid to the causative factors.
Some examples of this are hypertension or G.E.R.D.
medications used long term. There are also ADHD and
SEPTEMBER 2013
asthmatic medications utilized chronically with little
investigation or correction toward the etiological
elements that led to the current syndrome.
Historically and realistically, we are wonderfully made
and enabled with near countless abilities to compensate
for less than optimal nutritional intake, severe physical
injury, inadequate oxygen availability and chronic toxic
exposure, aswellas organ system malfunction. We
can have a multitude of maladies going on singularly or
simultaneously, especially on a chronic basis, with the
occasional OTC symptomrelieving medication as
deemed  . We are able to carry a
full load until we buckle at our knees and succumb to
our illness.
Holistically, through the DABCI model of chiropractic
care, we identify the organ systems involved, determine
the severity of dysfunction(s) present and correct the
maladies amendable to natural health care provision.
We make appropriate referrals as necessary, keeping
the patient fully informed of the working diagnosis and
the treatment protocol being prescribed.
Most
important are the reexaminations scheduled
throughout the prescribed treatment procedures to
assure both patient and practitioner of the efficiency of
our chosen protocol.
The DABCI system of health care is obviously the
superior choice, provided immediate death is not
imminent; however, as with many other truths
throughout history, the DABCI method of health care
provision is met with disdain and relegated not worthy
by those wielding the largest health care dollar axe; i.e.,
third party health care payers. This financial hardship
can be overcome by going to a cashforservice
payment system. The current diagnostic and procedural
codes are provided to each person we have the
privilege to serve, ensuring that they can file their claim
with their 3rd party payer. This keeps cost reasonable
and leads to the proper placement of anger. The
doctor/patient relationship can continue to focus on
health, while the haggling can be between the patient
and their insurance company as it should be.
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



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





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      


     
        


        

       
        
       
       
     

       
 trans      
      


The DietHeart Disease Hypothesis makes three pri
mary assumptions:
1.) Intake of saturated fat causes high total cholesterol
and lowdensity lipoproteins (LDLs)
2.) High total cholesterol and LDL are correlated with
heart disease
3.) High dietary intake of saturated fats is correlated
with high total cholesterol and LDL, and therefore,
causes heart disease.
While the first two correlative assumptions have
received wide support in the literature, recent
metaanalysis has concluded that dietary fat is not sig
nificantly correlated with coronary heart disease or car
diovascular disease.1
It is just as wellknown, yet less publicized, that satu
rated fat intake may also:
 Increase     or
"good" cholesterol.
 Decrease 
 Lower  , an independent
marker of heart disease risk.
 Improve the   , also a
strong independent marker of heart disease
risk.2,3,4,5,6,7,8
So while the last century of diet guidelines have fo
cused on the correlation of saturated fat, LDL's, and
Total Cholesterol with heart disease, eating a diet high
in saturated fats may actually be heart . How
so? While saturated fat intake undoubtedly increases
LDL's and Total Cholesterol; its beneficial effect on
other markers of heart disease may be more profound.
The  "beneficial" effect may go against everything
you have learned up to this point about saturated fats,
cholesterol, and their proposed links to heart disease.

The research community has largely ignored evidence
with regards to the potentially healthy effects of satu
rated fats on Lp(a), HDL, and the Total Choles
terol:HDL ratio.
Instead, much of the research has focused on saturated
fat intake and its association with high LDLs. Since
high LDLs are considered a risk factor for heart
disease, researchers have prematurely jumped to the
conclusion that saturated fats are to blame.
Our understanding of the connection between LDLs
and Heart Disease has also adapted. We now know that
different types of saturated fats and carbohydrates have
differential effects on .
Heart disease is not necessarily associated with total
LDLs, but it is definitely associated when there are nu
merous LDLs that are more "small and dense" in size.
The larger, "fluffier" LDLs may be heart protective.9
For instance, you may have high LDLs, but the major
ity of them may be of the protective, "large and fluffy"



Spring 2006
SEPTEMBER 2013

variety. On the flip side, you may have "normal" levels
of LDLs, but the majority of them could be of the risky,
"small and dense" variety.
It is these findings that may help explain why 50% of
136,905 individuals hospitalized with coronary heart
disease had "normal" levels of cholesterol, HDL, and
LDL at the time of their hospitalization.10
Findings now suggest that total cholesterol, LDLs, and
HDLs are not the most reliable indicators for heart
disease risk. LDL particle number is now accepted as
one of the strongest markers of heart disease risk, po
tentially even more than traditional markers like total
cholesterol and LDLs.11

As discussed, an increase in saturated fat intake
decreases the risk of heart disease; therefore, a decrease
in saturated fat intake should also be associated with an
increase in heart disease risk.
What does the research say about this?
In line with what you have been taught before, a
decrease in saturated fat intake is associated with a de
crease in LDL. But as you now know, the story does
not end there. Levels of healthy HDLs also decrease
and unhealthy Lp(a) levels increase.
When you choose to lower saturated fat intake, you
have to balance the "improvements" in LDL numbers
with the negative effects on levels of HDL and Lp(a).12
Clinicians and researchers alike are now challenged
with a broader picture of heart disease risk than what
we've been taught over the last century.
The American Heart Association still recommends a
diet that is comprised of only 7% or less calories from
saturated fat,13 and interestingly, it has not given clear
guidance as to what to substitute the saturated fats with.
Let's take a look at what happens when you substitute
saturated fat intake with polyunsaturated fats or carbo
hydrates.
    

The research community has focused on the substitu
tion of polyunsaturated fats (PUFAs) for saturated fats
as a means of reducing heart disease risk based on the
conventional dietheart disease hypothesis.
When you substitute PUFAs for saturated fats, heart
SEPTEMBER 2013
disease risk does decline, but it is impossible to tell if
the decline is because of adding more PUFAs to the
diet, consuming less saturated fats, the combination of
adding PUFAs and reducing saturated fats, or if the
correlation is due to other unknown variables not yet
considered.14,15,16
The underlying assumption appears to be that PUFAs
are associated with lower levels of LDLs and, there
fore, are correlated with less incidence of heart disease;
ultimately, researchers have generally concluded that
PUFAs are heart protective.
As we know from my earlier discussion however, the
widely accepted correlation between LDL levels and
heart disease is not cut and dry  yet researchers still
make the assumption that LDLs are bad, citing the
same correlative research articles over and over again.
Again, this is just a , and the potentially
healthy effects of dietary fats on HDL, Total Choles
terol:HDL and Lp(a) are still largely ignored.
Complicating matters more, a recent metaanalysis
reviewed that an increase in PUFA intake was associ
ated with a 13.3% reduction in Total Cholesterol as
expected; but the substitution of PUFAs for saturated
fats was also associated with an in mortality.
The authors proposed that the higher intake of PUFAs
was associated with increases in oxidized metabolites
(which make up most of the fatty acids in oxidized
LDL). Nevertheless, their findings went counter to
previous assumptions in the literature; and the authors
were careful to indicate that the trouble with PUFAs
may be when they are consumed in   
 rather than when found naturally in whole
foods such as nuts and vegetables.17 Excess dietary
polyunsaturated fat is nearly impossible to achieve with
a diet of whole foods.
It gets even trickier when you consider that oxidized
LDLs are an often proposed cause of atherosclerosis.
When you look at the composition of arterial plaques,
however, they are predominantly comprised of
oxidized polyunsaturated fats, not saturated fats.

Without clear guidance as to what to substitute
saturated fats with, most Americans have made up for
their saturated fat intakes with carbohydrates, espe
cially refined carbohydrates and added sugars.18
It is wellestablished that high intake of refined carbo
hydrates is associated with increased risk for type 2


diabetes and ischemic heart disease.19
hash browns, pepperoni pizza...
When you substitute carbohydrates for saturated fat,
little to no benefit is seen in reducing heart disease risk
unless the carbohydrates added are unrefined and have
a low glycemic index.20
When research subjects fill out food frequency ques
tionnaires, a check for "hamburger" is a check for satu
rated fat. But what do we eat hamburgers with? Typi
cally a white, refined carbohydrate bun, along with a
high sugar condiment like ketchup. Eating a hamburger
is not a simple categorization. The "white bread" and
"condiment" part tends to be ignored.
Additional analysis revealed that when saturated fat
intake was replaced with highcarbohydrate, high gly
cemic index foods, it was associated with a higher risk
of a heart attack. When the opposite occurred
(substituting saturated fats with low glycemic carbohy
drate foods)  it was associated with a lower risk of a
heart attack.21
Further, researchers suggest that even if we decided
that saturated fats are risky for heart disease, the effect
of high intake of refined carbohydrates may cause even
worse metabolic damage.22
So it would appear that more focus should be placed on
reducing refined carbohydrates, added sugar and proc
essed foods, and less focus should be placed on reduc
ing intake of dietary saturated fats.

There is no question that LDLs are associated with
higher levels of heart disease. But as we've established,
the story goes deeper when it comes to saturated fats
and cholesterol, LDLs, LDL size, LDL particle num
ber, HDLs, Lp(a), Total Cholesterol:HDL ratio, and
subsequent “net” risk for heart disease.
First, cholesterol intake actually has little to do with
cholesterol levels in the bloodstream.
As intake increases, the body's natural production of
cholesterol decreases. As the intake of cholesterol de
creases, the body's production ramps up, increasing
levels. Paradoxically, you can have elevated, poten
tially harmful levels of cholesterol due to not eating
enough cholesterol.
The research now suggests that saturated fat intake may
have a greater beneficial effect on HDLs, Total Choles
terol:HDL, Lp(a), and LDL Particle Size and Number.
When you substitute saturated fat intake with refined
carbohydrates and other processed oils high in PUFAs,
you may actually be increasing your health risk.

When you think of typical American foods, what do
you think of? Hamburgers and french fries, bacon and


Spring 2006
Additionally, not all studies control for processed vs.
unprocessed meat as sources of saturated fat.
One study did show an association between processed
and unprocessed meat with stroke mortality, but the
analysis was still assessed by an FFQ.23
FFQ's do accurately reflect saturated fat intake, but the
important grey areas are not taken into account.
An additional study from the  made
a really interesting observation, however.
The researchers looked at total cheese intake and the
use of butter in cooking (high sources of saturated fat).
They failed to find an association with heart attacks for
both, but that was not the interesting part. When they
controlled "how" the butter was consumed, they found
when it was consumed on bread, it was  asso
ciated with heart attack risk; when consumed alone, no
correlation was found.24

In April 2013, the NY Times and other publications
sent the media on a fury announcing a new proposed
link between red meat and heart disease.25
The researchers found that those eating red meat were
metabolizing its carnitine content into trimethylamine
Noxide (TMAO). The study also showed a correlation
between TMAO levels and heart disease risk.26 The
media concluded that a new link between heart disease
and red meat had been discovered, yet flaws in the as
sociation surfaced.
One of the highest natural sources of TMAO is fish.
TMAO is actually the chemical that gives fish its
"fishy" smell. Fish intake has largely been associated
with heart disease protection. That was the first glaring
problem with their analysis.
The second problem took a little deeper analysis. The
mechanism for the conversion of carnitine to TMAO

SEPTEMBER 2013

proposed by the researchers was the overgrowth of cer
tain species of bacteria in the gut such as .
What might increase the level of  bacteria in
the gut? A highgrain diet has been proposed as root
cause for the overgrowth.27
The media could have just as easily reported that a new
link was discovered between heart disease and grains,
but the bias toward red meat and saturated fat was
maintained in the coverage.

 fatty acids (TFAs) comprise 25% of total fat in
ruminant fats (from cows, goats, sheep, etc.) but com
prises 5060% of total fat in partially hydrogenated
vegetable oils.
Naturally found TFAs differ from those found in par
tially hydrogenated vegetable oils because of the place
ment of the double bonds. The most commonnaturally
occurring TFA is vaccenic acid and is the precur
sor to conjugated linoleic acid (CLA or alpharumenic
acid).28
While CLA may still have a deleterious effect on lipid
profiles like other  fatty acids,29 it may also offer
multiple beneficial effects for fat and bone metabolism,
immune function, as well as risk of cardiovascular dis
ease and cancer.30 It is generally considered a healthy
fat when compared to the TFAs found in partially hy
drogenated vegetable oils, but the research is still
young.
Dietary TFAs raise LDL cholesterol and total choles
terol, and lower HDL cholesterol31 TFAs have also
been reported to increase the stickiness of platelets in
the blood32 as well as levels of Lp(a).33
The news is troublesome as up to 70% of the vegetable
oils used in foods like crackers, cookies, pastries,
cakes, snack chips, candies and fried foods are partially
hydrogenated. Because of this, it is very difficult to
assess TFA intakes among populations.28
Food companies are only required to list fat (TF)
on the label if the TF content is more than 2g per serv
ing. If the label reads partially hydrogenated fat in the
ingredient list, but 0g of total TF, it is likely that the
product contains TF, but less than 2g per serving.
Estimates for American intake of TFAs range as high
as 38.7g/day.28
Other possible adverse effects of TFAs include, but are
not limited to:




Cognitive decline
Metabolic syndrome
Infertility
Compromised fetal development 33,34
While TFAs naturally found in milk, cheese, and rumi
nant meats may be healthy, one might consider staying
away from pastries, cookies, snacks, fried foods, mar
garine, and other processed foods containing unnatural
partially hydrogenated vegetable oils.
     

1.) Saturated Fat and Cholesterol may not cause heart
disease. They may actually be heart protective.
2.) Be cautious of processed polyunsaturated fat
(PUFA) oils that have been used to substitute satu
rated fats in our diet. PUFAbased oils may in
crease risk of vessel damage from oxidized LDLs.
Be particularly careful when these oils are:
 Cooked at high heat
 Stored for long periods of time
 Exposed to long periods of air/light
 Partially hydrogenated/hydrogenated
 Consumed with refined, processed, high
glycemic index carbohydrates
3.) Stay away from refined, processed, high glycemic
index carbohydrates. In particular, white breads,
refined grains and added sugars.
4.) Stay away fromTFAs found in margarine, pastries/
baked goods, and partially hydrogenated/
hydrogenated oils. Some natural  fats such as
conjugated linoleic acid (CLA) may offer health
benefits.

      
      
    

     
         
       
       
      

       

     

SEPTEMBER 2013

     



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rated fat in place of saturated fat: A systematic re
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view and metaanalysis of randomized controlled
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J, Tiǿnneland A, Schmidt EB, Overvad K. Intake
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portance of the glycemic index. Am J Clin Nutr.
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fatty acids on highdensity and lowdensity lipo
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32) Wahle KW, Peacock LI, Taylor A, Earl CR. Effect
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34) Bhardwaj S, Passi SJ, Misra A. Overview of 
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SEPTEMBER 2013
35) Weinberg SL. The dietheart hypothesis: a critique.
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
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
Recently I met a Harvard trained Cardiologist in a so
cial venue. He told me that only double blind studies
were of value. This led me to my compulsive obsessive
tendency to think, and here are some of the thoughts
that emerged from the occasion.
First, let me say that it appears to a country boy, such
as I, that this is an elitist viewpoint. How much of the
knowledge in this world has no such double blind his
tory? How many double blind studies were done be
fore the first mitral valve was replaced, and since the
answer is none, shouldn’t we stop mitral valve replace
ments until such double blind evidence can elucidate
the efficacy of such procedures?
Double blind has been used as a whipping post for
those of us who use natural medicine because so much
of what we do is based on clinical evidence. If double
blind studies are the only good way to obtain valuable
information and if clinical studies count for nothing,
won’t we have to put most medical procedures on
hold?
Many things do not readily lend themselves to the dou
ble blind protocol. Manual manipulation is one of these
things. Does this mean that we must forego all manual
manipulation until such studies can be done?
This is not to say that double blind studies are not use
ful and desirable whenever they can be applied. The
reason this method has been successful in the testing of
many kinds of medicine is that the process is beyond
partisan control, and the results are free of interpreta
tive bias. Double blind works well when the response is
linear and the process being examined is completed
within the sixmonth test period. Many drugs do lend
themselves to the double blind format, but even they
have some weaknesses. In many instances, the medica
tion in question is compared to a placebo. A placebo,
by definition, does absolutely nothing. How difficult is
it to compete with nothing and just how good is the
drug that has won such a contest? This leads us to an
other obvious fault.


Spring 2006
I would ask Dr. Double Blind how he knows beyond
any shadow of a doubt that his placebo has no action
whatsoever. Ask any person you meet to give you an
example of a placebo and at least some of them will say
“a sugar pill.” You and I know that sugar has very sig
nificant effects on the human body and many of them
are very detrimental.
Of course Dr. Double Blind knows that, so if he were
developing a research study, he would not pick sugar
for his placebo. What would he pick and how would he
determine that it has no long term physiological
effects?
Even when we have double blind studies, it sometimes
isn’t enough; there can be other factors. Was the study
done at a university? Was the researcher a famous sci
entist? What are his credentials? Was this study pub
lished in a peer reviewed journal? Was the study done
in the United States of America? Does the last question
imply that we can’t learn from third world researches,
but they have to learn from us? Oh my God, now I am
starting to sound elitist!
So let’s give Dr. Double Blind the criteria that he re
quires. Let’s say it is a double blind study, done at a
major university by a Nobel laureate and published in a
peer reviewed paper such as JAMA. So if the therapies
allopathic doctors are using are sacrosanct because they
are based on the gold standard of research, why do we
keep getting reports, from such sources as the Journal
of the American Medical Association, among others,
that adverse drug reactions are one of, if not the leading
cause of death in this country?
In his book, The New Arthritis Breakthrough, The Only
Medical Therapy Clinically Proven to Produce Long
Term Improvement and Remission, Henry Scammell
says the following: “
        
        


      

         


      

”
     

Bruce Pomeranz M.D., Ph.D., from the University of
Toronto analyzed 39 studies of Adverse Drug Reac

SEPTEMBER 2013

tions in the United States to estimate the incidence of
serious and fatal reactions in hospitalized patients. The
results were that 106,000 deaths each year are caused
by patented medicines. It did not include outpatients,
mistakes, or accidents; if it had, the number would have
been much larger. This study was published in the
Journal of the American Medical Association, April
15th, 1998. I wonder how consoling it is to the families
of the deceased to learn that all of the drugs involved
were approved by double blind studies.
Barbara Starfield M.D., of Johns Hopkins School of
Hygiene and Public Health, confirmed the above report
and shed light that the numbers were actually much
higher. This was reported in JAMA, July 26th, 2000.
284(4):21845. To add interest to the facts, Dr. Star
field passed away and the cause of death has been at
tributed to her medical care. Implicated is the com
monly prescribed and widely advertised drug Plavix, a
drug proven by double blind studies and approved by
the FDA.

No matter how double blind the study is, if the animal
tested is inappropriate for the test, the results will be
meaningless. A perfect example of this is using rabbits
for cholesterol studies. Cholesterol does not exist in
plants but only in animals. Rabbits don’t eat animals.
To use rabbits in cholesterol studies, one has to force
feed cholesterol because they won’t eat it otherwise.
Since rabbits are not designed to handle cholesterol in
their diet, the results are meaningless if you are trying
to correlate them with humans.


The heart of many research papers lies in the statistical
details. It has been said that Statistics are like a bikini;
what it reveals is interesting but what it conceals is vi
tal. The difference frequently lies in whether the results
are a relative risk or an absolute risk. Once it is known
whether the researcher is using relative or absolute risk
factors, one needs to look at the NNT factor (number
needed to treat). How many people must be treated be
fore one person has the desired effect? It is not possible
to cover these factors thoroughly in this paper, but
every doctor should make sure that he or she under
stands these concepts. Pharmaceutical companies have
repeatedly proven that they will tinker with the statis
tics to get their research projects approved. Each of us
must make sure that we understand the game when we
read these research papers. You can go to Biotic’s
Tuesday Minute (www.tuesdayminute.net) and find an
introduction of the answers if you want to refresh your
understanding of how to interpret a research paper.
SEPTEMBER 2013

Once you have done your double blind study, or any
other study, you must interpret the data. It is not diffi
cult to find relationships between two parameters. The
problem starts when one wants to define the cause and
the effect. Just because there is a correlation between
cholesterol and heart disease, does not guarantee that
increased cholesterol causes heart disease. Could it be
possible that heart disease causes cholesterol to in
crease? An example of this logic can be illustrated by
the fact that every time you see a house on fire, you see
a fire truck. This is not proof that fire trucks are a major
cause of house fires. As wonderful as the double blind
design is, it contributes very little to the interpretation
of cause and effect.

Last, but not least, is the question of integrity. We
Americans, and probably the rest of the world, have
come to place research on a pedestal. It is one of the
last refuges where we can place trust. When I was
young, there were many things that we believed in and
trusted: policemen, teachers, clergy, doctors, almost
anyone in authority and most of all, an almighty God.
For many people all of the above are no longer a court
of comfort when we need one. Until recently, research
has remained something that most people could believe
in.
It gives me no joy to report that people are now begin
ning to lose this refuge. The government has brought
many law suits against pharmaceutical companies in
recent years. Fraud, inappropriate charges to Medicare,
and just general crookedness that would land you or me
in jail have become commonplace.
Pharmaceutical companies are now financing most of
the drug research that is being done. They are also fi
nancing the FDA. When you are buying research, you
can bet that you can get what you want. They are using
research, usually double blinded, to legitimize patent
medicines which are promoted to be health promoting
but are killing us by the thousands.
The latest settlement that I am aware of was against
Glaxosmithkline for three billion dollars in 2012.
These companies are making so much money that they
can afford to pay billions of dollars in fines to the gov
ernment and continue to do business as usual. They
consider it just another cost of doing business, but it is
also a money making plan for both the drug companies
and the government.
This article was not intended to promote a moral or
religious message; however, I feel inclined to conclude
with the thought that if double blind studies are the
only thing that you have left to believe in, may God
save us all. 

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 

Dr. Harper goes further, addressing the same questions
Dr. Feinberg and thousands of scientists are asking
today about epigenetic control of the cell. Dr. Feinberg
writes:


1) What is the gross reaction of the organism to
changes in the environment?
2) What is the analysis of the simpler reaction of
which the gross reaction is composed?
3) What is the assignment of each cell, its part in
every reaction, and how is it controlled normally?
In    , Dr. William Harper
wrote, “We might define health as controlled irritation
of the nervous system that leads to adaptation; that is;
some  demand starts the process that
causes innate to direct the impulses which control
function for .”1
Andy Feinberg, MD, MPH, professor of molecular
medicine and director for Epigenetics at Hopkins’
Institute for Basic Biomedical Sciences found
reversible tagging of methylation patterns of genes
respond to changes in the  controlling the
 of the genes. Epigenetic scientists including Dr.
Feinberg have determined that “
      
2
Dr. Harper’s book was an explanation of chiropractic
theory as promulgated by DD Palmer. The essence of
Dr. Harper’s treatise resulted in a formula for
chiropractic theory. It is important to remember Dr.
Palmer was searching for the universal formula for
disease when he “developed” chiropractic. Here is the
formula for the chiropractic theory:
Disease= Environment=Health
Resistance
When you study this formula in its entirety you will
understand that this new science of epigenetics isn’t
new at all. In fact, you will see that epigenetics explains
what Drs. Harper and Palmer were communicating
about health and disease, starting in 1895 and expanded
in the 1960s. Dr. Harper called chiropractic the science
of existence. This is no simple concept and requires
serious study of every branch of science.
There is a critical bit of information here. If missed,
you will miss the entire concept of this formula. Dr.
Harper defined environment as: “The totality of
influences acting upon the organism 
” Internal is intracellular and external is
extracellular matrix, according to Harper.1
SEPTEMBER 2013
“If we could answer the following questions for every
system, organ, and cell in the body, there would be no
questions that we could not answer.”
Dr. Harper addressed these questions in 1964, minus
current questions such as; does the attachment of the
DNMT3A epigenome to the Cp6/DNA cause the gene
to express a certain disease or condition? Dr. Harper
said, “this knowledge would explain how and why
every symptom or abnormal function occurs. This is
diagnosis. This concept is peculiar to chiropractic. Let
us use it, even if it means discarding ideologies that
have no basis in fact.”1
Chiropractic theory adds another layer to the current
theory of epigenetic control of the cell. Chiropractic
theory states that this expression of the cell is
coordinated through the nervous system. In fact, Dr.
Harper defines the line itself between environment and
resistance as the nervous system in the chiropractic
formula.1 Epigenetics struggles with this and in some
of the literature even calls it . Most of the
epigenetic scientists drop the idea at this point for the
fear of being associated with the untouchable idea of
including theology in science.
Epigenetics is the expression of the genes in response
to the internal and external environment of the cell.
This expression can be short term or long term,
abnormal or normal, but it is in response to changes in
the cell environment. This is a new concept in medicine
and is taking the scientific world by storm. This is right
out of Dr. Harper’s book explaining DD Palmers
principles.
It is becoming clear that the gene itself plays only a
partial role in health and disease. It isn’t the controlling
factor as current genetic theory holds. It is the
epigenetic control of the gene that determines the
expression of the gene. Cellular biologists and
epigenetic geneticists understand that methylation of
the gene is a key to epigenetic control. This control is a


disease.”3 Dr. Harper, using DD Palmers ideas,
theorized that these neurological reflections from an
altered cellular environment, and the reaction of the
cells to this abnormal environmental stress, resulted in
the chiropractic subluxation complex. He even defined
the nervous system as the mediator in his formula for
disease and health.
result of the cell responding to a changing environment
of the body either macro, micro, internal or external.
Dr. Harper elucidated bacterial, viral, chemical,
physical, electrical, psychic factors that are responsible
for abnormal cellular environments and that the
nervous system reflects those changes physically in the
body and the spine. He also postulated that the nervous
system ultimately controls the reaction of the body to
the abnormal cellular and extracellular environment
and treatment of the nervous system is vitally
important. He didn’t know about epigenomes, histones,
and methylation of genes and other new discoveries
that control the cell’s reaction to its environment but I
would imagine he would be very excited to read the
new research.
Dr. Harper, in light of this new information, might say
that any stress; chemical, structural, or electrical, can
result in the changing of the cellular environment;
resulting in epigenetic control changes in the
expression of the genes. Which, in turn, is reflected
through the nervous system and may manifest in a
biomechanical alteration of the body.
The scientific world has arrived in 2013. Chiropractic
started in 1895. Today’s modern theory of disease is
Chiropractic 101, right out of Dr. David Harpers
textbook, explaining DD Palmers theory of
chiropractic.
Today, we know about endocrine disruptors, receptor
disruptors, genetically altered foods and micro dose
chemicals, xenobiotics and drugs that change cellular
environment causing an abnormal expression of the
genes and disease. Chiropractic theory states, any
physical, chemical, psychic factor that causes irritation
of the nervous system results in functional pathology.


1)
A recent study in stated, “It is
becoming clear that all complex diseases are the result
of geneenvironment interactions and that epigenetic
modification of gene function during critical periods in
development plays a critical role in the etiology of
2)
3)

Harper, William David. ; 

Seabrook, TX: Texas Chiropractic College,
1974.
Science News, , Sept. 16, 2012,
page 1.
, Seabrook, TX: Texas
Chiropractic College 102 (2) (2008), pp. 7681.

 
Board testing for the DABCI Diplomate Degree is scheduled for
October 1920, 2013 and April 2627, 2014


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
Spring 2006
SEPTEMBER 2013
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INNOVATIVE ALLERGY AND IMMUNOLOGY SERVICES
Toward Better
Health…
» Inhalant Allergy
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» Intestinal Bacteria
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   
 
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 
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
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 

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 

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 

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 


 
 
*Every attempt is made to offer these seminars as publicized; however, we reserve the right to adjust seminar locations, dates, times, etc. due to circumstances beyond our
control. Program continuation is contingent upon adequate attendance. No audio or video tape recorders are allowed, and no portion of the seminar may be reproduced in any man
ner without expressed written consent. Preregistration is required. ProHealth Seminars shall not be held responsible for any expenses incurred by registrar if a program must be
altered and/or cancelled. Seminar fee is nonrefundable. Credit cards will not be billed until we have the minimum number of attendees (20). If the Doctor is unable to attend after
preregistering and payment has been received, the seminar fee will be transferred to another Seminar (attended within 12 months of the original seminar) for an administration fee
of $25. Any seminar fee not transferred and used within 12 months will be forfeited.
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
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 
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*Every attempt is made to offer these seminars as publicized; however, we reserve the right to adjust seminar locations, dates, times, etc. due to circumstances beyond our
control. Program continuation is contingent upon adequate attendance. No audio or video tape recorders are allowed, and no portion of the seminar may be reproduced in any man
ner without expressed written consent. Preregistration is required. Pro Health Seminars shall not be held responsible for any expenses incurred by registrar if a program must be
altered and/or cancelled. Seminar fee is nonrefundable. Credit cards will not be billed until we have the minimum number of attendees (20). If the Doctor is unable to attend after
preregistering and payment has been received, the seminar fee will be transferred to another Seminar (attended within 12 months of the original seminar) for an administration fee
of $25. Any seminar fee not transferred and used within 12 months will be forfeited.
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
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
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

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

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
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Pro Soothe Stress Cream
Maca Extract, Glutamine, 7-Keto DHEA, Pregnenolone
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







:
It is becoming more evident that hormone balance is
not only vital to a healthy lifestyle, but necessary for
health, vitality and longevity. Men and women alike
suffer from hormone imbalances now more than ever,
due to stress, environment, poor diet, toxins and life
style choices. As a health care provider, it is necessary
to understand how hormones affect the health of the
patient and their quality of life.
“Estrogen dominance,” which is another term for
progesterone deficiency (coined by the late Dr. John
Lee (MD)),1 is very common. Progesterone deficiency
is not only due to excess exposure to estrogenic sub
stances like xenoestrogens, but due to long term stress
and its effect on the adrenals, which is compounded by
the body’s inability to rid itself of excess hormones.
Stress can be either emotional or physiological; pain,
lack of sleep, blood sugar imbalances, digestive weak
ness and toxins.
In women, progesterone deficiency is a key factor in
PMS management and menopausal symptoms, as well
as, estrogen receptor positive (ER+) breast cancers.
Typically, allopathic medicine leans on synthetic
hormones to manage symptoms related to hormone
imbalances. Synthetic hormones such as birth control
or synthetic hormone replacement therapy (HRT) are
not readily excreted from the body as natural hormones
should be. This can cause long term estrogen domi
nance, and/or exacerbate existing symptoms of mood,
weight, sleep or menstrual irregularity.2
SEPTEMBER 2013
Balance is the key to hormonal health. Estrogen recep
tors can become less sensitive when progesterone is
low for long periods of time. Supplementing with natu
ral progesterone balances the ratio of estrogen to
progesterone and increases the sensitivity of estrogen
receptors. 1
In conclusion, this study shows that topical trans
dermal progesterone therapy does raise progesterone
levels in the body and can be a viable way to relieve
symptoms of PMS and menopause without the danger
ous side effects reported from utilizing synthetic hor
mone replacement therapy or birth control.


This clinical study was performed to determine the
effectiveness of a natural transdermal progesterone
cream in raising progesterone levels in women between
the ages of 3555, who are either perimenopausal or
full menopausal. Participants were not involved in any
other health regimens, nor were they under the care of
any physician or had consumed hormone supplements
or prescriptions in the last 6 months. Each participant
submitted a saliva sample before and after the study to
confirm progesterone levels at that time. Symptom
questionnaires and vitals were collected every two
weeks for the majority of participants, with a couple
who only provided a before and after symptom ques
tionnaire. Only the before and after symptom question
naires were used for chart two on page 123.
The supplements used for this study were provided by
Professional Health Products (PHP) and are; Transder
mal PRO SOOTHE STRESS ADRENAL SUPPORT
CREAM with the ingredients: Water, Maca Extract,
Glycerine, Octyl Salyclate, Glycerol Monolaurate,
Glycerol Stearate/PEG 100, Cetearyl, Alcohol/Cocoa
Glucoside, Caprillic Triglyceride, Safflower Oil, Puri
fied Lecithin, 7Keto DHEA, DiIndolylmethane, Thioc
tic Acid, Vitamin E, Glutamine, Pregnenolone, Cellu
lose Gum, Peg 30, Glycerol Cocoate Phenoxyethanol,
and Methisothiazolinone. This cream aims to balance
the adrenal glands and provide ingredients to support
the adrenals as they produce cortisol so as not to con
vert progesterone to cortisol.
The progesterone cream used was the Transdermal
PRO FEMME SUPPORT + FEMALE SUPPORT
CREAM with the ingredients: Water, Wild Yam Ex
tract, Black Cohosh Extract, Glycerine, Octyl Salyclate,
Glycerol Monolaurate, Glucerol Stearate/PEG 100,
Cetearyl Alcohol/Cocoa Glucoside, Progesterone,


Capryllic/Capric Tryglyceride, Dafflower Oil, 7Keto
DHEA, Purified Lecithin, Evening Primrose Oil. DI
Indolylmethane, Cellulose Gum, PEG30 Glyceryl Co
coate, Phenoxyethanol, Methisothiazolinone. This
cream was used to raise natural progesterone levels in
the body. Dosage was one metered pump AM and one
metered pump PM of Pro Femme Support + Female
Support and one metered pump AM of Pro Soothe
Stress Adrenal Support each day for about 56 weeks or
until the Pro Femme cream was gone.



When addressing female hormone levels it is important
to consider the reason(s) for imbalance. Even women
who do not display troublesome symptoms may experi
ence symptoms which a well trained health provider
can detect. Patients with liver congestion, poor sugar
management, high stress, etc., are candidates for hor
mone imbalances and conditions such as; PCOS,
weight gain, memory problems, migraines and other
symptoms (which many people assume are “normal”)
can be related to hormone imbalance as well. Adequate
progesterone is necessary for regular menses, preg
nancy, regulating PMS, fat burning, reducing water
retention, and protecting against certain female–related
cancers as well as preventing the conversion of testos
terone to DHT (dihydrotesterone), which prevents acne,
facial and body hair, thinning of the hair and oily skin.
Proper levels of progesterone also ease the transition
into menopause, and reduce the symptoms that many
American women experience. Menopause is not typi
cally as serious of a condition for women in other coun
tries where processed foods and a high stress lifestyle
are not as common.
In times of stress, the adrenals “steal” progesterone to
make additional cortisol.3,4 In a perfect world, addi
tional cortisol is only needed to get through a period of
stress, but in our highstress world many people never
get through the short burst of stress. It becomes a con
stant part of their lives which depletes progesterone
long term. Treating low progesterone without address
ing adrenal insufficiency can be very difficult. In order
to raise progesterone, the adrenal issue must also be
addressed. This may include lifestyle modifications and
overall health evaluation. See for more infor
mation on these pathways.3 
During this study each participant was provided with an
adrenal support cream as well as a natural progesterone
cream to support the adrenals and aid in raising proges
terone levels.


When taken in tablet form, any supplement (including
natural progesterone) is often not fully absorbed due to
a less than efficient digestive system. Topical creams
are a more reliable option and allow progesterone and
other nutrients to be delivered directly to the blood
stream, bypassing the digestive system. Many topical
creams require the patient to apply them to fatty areas
and to rotate the area of application daily to avoid build
up in the tissues. Build up over time will drastically
reduce the effectiveness of the cream, which will allow
the “estrogen dominance” symptoms to return, because
excess progesterone produces the same symptoms as
too little progesterone. This is due to progesterone
receptor sites becoming “numb” to the hormone, which
is similar to the body’s response with insulin resistance.
It is not necessary to rotate a true transdermal cream
that uses a liposomal delivery system because it does
not build up in the tissue. These creams are applied to
thin skinned areas and require no rotation.5
For this study, women were advised to collect the final
saliva sample 36 hours after the last dose of cream. Due
to the nature of this study other health concerns were
not factored in. It is unknown which of these women
suffered adrenal insufficiency or had other health con
cerns that may have contributed to their final results.
The goal of the study was to prove the liposomal deliv
ery of topical progesterone would raise progesterone in
this particular group of women.


Saliva testing was utilized in this study because proges
terone is readily measured by saliva testing. Serum
levels of progesterone are not bioavailable, therefore
serum testing is not accurate for this purpose.6
Healthy levels of progesterone measured as pg/ml are
300500 in saliva, but during supplementation due to
the necessity to saturate the tissues with the proper
amount of progesterone, levels above that are accept
able. Variation of progesterone in the body is typically
well tolerated up to 1500 pg/ml.6


Based on the saliva results we did find that progester
one levels were raised in all of the women who partici
pated. Some were small increases and others were
much more dramatic. This suggests that the overall
health of the individual is a factor and should be taken



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
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
into consideration when creating an individualized pro
gram. (See Chart 1: page 123.)
Of the 13 participants who completed the study, 4 of
them were still cycling, 9 were menopausal and 4 of
those reported surgically induced menopause.
Out of the 13 participants; 5 participants showed an im
provement in hot flashes, 1 noted no hot flashes prior to
the study, 6 noted an improvement in insomnia, with
two stating they had no trouble sleeping prior, 7 showed
an improvement in energy, night sweats and libido with
2 showing none of those symptoms prior, and with 5
stating no night sweats prior to the study, 8 participants
showed an improvement in depression and fluid reten
tion, with 2 stating they had no previous symptoms of
depression and 4 stating that fluid retention was not a
problem, 9 showed an improvement in anxiety with one
stating no anxiety prior to the study, and 11 stated that
their mind raced less, with one person stating she had no
mind racing issues previously.
Over half of the participants showed an improvement of
25% or more in all symptoms recorded except hot


Spring 2006
flashes and 5 of them reported an improvement. This
study shows that all women who participated experi
enced some increase in progesterone by saliva testing.
(See Chart 2: page 123.)

1) Lee, John.      
(2004)
2) Cutillo, Dawn M. . (2012) Print.
p. 2627
3) William G Timmins    ,
(2011) p. 49
4) Hormones and Chronic Stress   
 Web; http://biohealthlab.com/testmenu/
hormones/hormonesandchronicstress/ 8/2013
5) Cutillo, Dawn M.   .(2012)
Print. p. 158
6) Saliva vs. Serum or Plasma Testing for Progester
one        Web
w w w . j o h n l e e m d . c o m/ s t o r e /
saliva_serum.html#absorb 8/2013

SEPTEMBER 2013

Chart
1

EƵŵďĞƌŽĨWĂƌƚŝĐŝƉĂŶƚƐtŚŽ^Ăǁ/ŵƉƌŽǀĞŵĞŶƚ
ĂŶĚWĞƌĐĞŶƚĂŐĞŽĨ/ŵƉƌŽǀĞŵĞŶƚ;ϭϯWĂƌƚŝĐŝƉĂŶƚƐͿ
ϴ
ϳ
ϲ
ϱ
ϰ
ϯ
Ϯ
ϭ
Ϭ
ŶdžŝĞƚLJ ĞƉƌĞƐƐŝŽŶ DŝŶĚZĂĐĞƐ
EŽ^LJŵƉƚŽŵƐ
&ůƵŝĚ >ŽǁŶĞƌŐLJ
ZĞƚĞŶƚŝŽŶ
Ϯϱй/ŵƉƌŽǀĞŵĞŶƚ
EŝŐŚƚ ,Žƚ&ůĂƐŚĞƐ
/ŶƐŽŵŶŝĂ
^ǁĞĂƚƐ
ϱϬй/ŵƉƌŽǀĞŵĞŶƚ
>ŝďŝĚŽ
ϳϱй/ŵƉƌŽǀĞŵĞŶƚ
2
Chart
Before and After Progesterone Level
SEPTEMBER 2013









, DC
The most powerful cancer fighter ever discovered is
naturally occurring vitamin D. The proof is in black
and white:
       
   


        

       


 
       

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  
        


     

sometimes free substance like vitamin D could reduce
national cancer rates by 75 percent.
This is a shame, but doesn’t surprise me… Our leaders
in medicine have sought to make us a nation dependent
on drugs and sought to suppress natural solutions even
if proven to actually work. Wow! That should make
you just as mad as it makes me!
Aside from cancer prevention, vitamin D cools the fire
of inflammation throughout the body. Less
inflammation means stronger, painfree joints and a
healthier heart. Vitamin D also:







Enhances mood
Boosts your immune system
Prevents bone and muscle weakness
Dramatically lowers risk of heart disease
Prevents diabetes
Fights arthritis, pain and inflammation
Helps prevent Parkinson’s disease and multiple
sclerosis
What’s astonishing is that the power of this simple
nutrient has been in plain sight for years. Study after
study has proven just how vital vitamin D is in
combating a host of ailments. Yet the good news still
hasn’t spread. It’s time to boost your vitamin D.
The easiest, safest and cheapest way (it’s free) is to
increase the amount of vitamin D the body produces
through regular exposure to sunlight. However,
depending where a patient lives, this is not always
possible. In that case, you can also take vitamin D as a
supplement. I recommend 5,000 IU every day.
Don’t rely on your multivitamin to give you all the
vitamin D you need, even if it does have D3. It’s a
good start, but most still only have around 400 IU.

Dr. Elena M. Morreale, DC, is a Doctor of Chiropractic
and a Bioenergetic Practitioner. She has been in
practice in Tampa, Florida since 1997. Graduating
from the State University at Buffalo, New York in 1990
with a B.S. degree in Medical Technology. She worked
at Mt. St. Mary’s Hospital in Lewiston, New York
from 19881993 as a Medical Technologist doing
laboratory and pathology analysis and diagnosis on
blood, urine, stool and body tissues. She is a skilled
practitioner with advanced EAV/EDS training in Bio
energetics which represents a new science able to
generate greater diagnostic and treatment pre
cision. She has taken numerous classes in Holistic

In fact, the Canadian Cancer Society contends that a
simple supplement of 1,000 international units of
vitamin D per day can reduce the risk of various
cancers by as much as 60 percent. This is one of the
most important findings in modern medical history:
Vitamin D is the best current hope for preventing
cancer. Period.
So why don’t we hear more about this stunningly
important news? Modern medicine seems to have no
interest in natural treatments and preventions for
cancer. Our FDA is more intent on prosecuting doctors
who stray from drug treatments than disseminating the
truth about natural cures. Even when a simple, and


Spring 2006

SEPTEMBER 2013

Medicines such as bioenergetics, homeopathy, herbals,
nutritional supplementation, energy work, emotional
stress integration and muscle testing.
She has
combined chiropractic care, nutritional supple
mentation and homeopathy for a complete holistic
health care practice. Her main goal is to find the
underlying cause of her patients problem and to tailor
an individual treatment plan. Dr. Morreale brings the
latest in alternative therapies with long lasting solutions
for health and longevity. She focuses on
helping people to heal themselves and regain their
health.


1) OrdóñezMena J, Schöttker B, Haug U, Müller H, Köhrle
Ortho Biotic
J, Schomburg L, Holleczek B, Brenner H. "Serum 25
hydroxyvitamin D and cancer risk in older adults: results
from a large german prospective cohort study." 
 2013 May;22(5):90516.
2) Grant, W.B. et al, “The association of solar ultraviolet B
(UVB) with reducing risk of cancer: multifactorial
ecologic analysis of geographic variation in ageadjusted
cancer mortality rates,”   2006;
26:26872700
3) Lappe, J.M., et al, “Vitamin D and calcium
supplementation reduces cancer risk: results of a
randomized trial,”     June 2007;85
(6):158691
4) Tuohimaa, P., et al, “Does solar exposure, as indicated by
the nonmelanoma skin cancers, protect from solid
cancers: vitamin D as a possible explanation,”  
 July 2007;43(11):170112 
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


 
 
 
 
 
 



SEPTEMBER 2013

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

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





A continually contracting muscle requires specific
nutrients and oxygen to sustain continued use. A lack
of nutrient supply to a muscle tendon region will lead
to overuse sports injuries. Therefore, let’s ensure that
athletes get a healthy nutrient supply to the body
through proper diet and supplementation that will assist
the body with its natural function and repair processes.
My take on why we should use supplementation is to
aid in the healing of sports injuries.
We need a simple and effective way to educate athletes
on the importance of approaching their healthy process
in 3 distinct phases:
 – Acute phase  first 72 hours of injury
  – Subacute (healing phase) – day 4 through 8
weeks
 – Wellness/prevention phase
The symptoms in the acute phase include acute muscle/
joint pain, and sprain/injury due to trauma or repetitive
motion. There will be visual swelling, inflammation
and spasm in the surrounding tissues. Additionally,
there will be a loss of, or decreased, function. The
objective of supplementation should be to aid in
managing pain, reducing swelling, relax tight muscles,
and use rehab strategies to restore motion.

Nutrients to address swelling – trypsin, chymotrypsin,
bromelain, which are proteolytic enzymes known to
reduce swelling and aid in pain and inflammation
reduction. These enzymes must be consumed on an
empty stomach for full effectiveness. Ginger, turmeric,
and boswelia are found to provide relief to pain and
inflammation without causing any sideeffects to the
athlete’s stomach. Calcium and magnesium are great
additions to address muscle tissue. Calcium works to
relieve spasms, and magnesium promotes muscle
relaxation. Normally,  to  is in a 2:1
ratio. However, for muscle injury, consume 
to  in a 2:1 ratio.


Spring 2006
   include continued joint or muscle
pain, visual inflammation surrounding the injured area
may still be present, and range of motion may still be
compromised. Tissue repair and remodeling occurs in
this phase! The objective of supplementation should be
to initiate softtissue support by modulating the
enzymes matrix matelloproteinases, help promote
repair, and remodeling of connective tissue in the
injured area.
Matrix metalloproteinases (MMPs) are our own
enzymes that are released at the time of injury.
Unfortunately, excessive release of MMPs can damage
healthy tendon collagen and connective tissue in the
injured area. This excessive release aids in the
degradation of healthy tissues. Therefore, if we
modulate the expression of these enzymes we are able
to support healthy remodeling of the connective tissue
in the area. Thiaa (which is a selective kinase response
modulator from certain plantbased components that
positively influences numerous aspects of good health),
berberine, selenium and folic acid are nutrients that
impact matrix metalloproteinases.
To support the growth and construction of connective
tissues, the following nutrients are suggested – glycine,
proline, lysine, vitamin C, B6, B5, Ltaurine, and silica.
I would recommend glucosamine and chondroitin
sulfate with MSM to aid in joint stability.
   are to achieve optimal tissue
remodeling and support wellness/prevention, also to
reduce the risk of reinjury and degeneration by
installing what I call my foundation nutrition:
1) Phytomulti – multivitamin/mineral complex with
additional phytonutrients
2) Omega3 fatty acids – to aid in the reduction of
inflammation (2 grams of EPA/DHA)
3) Vitamin D aids in the healing of sports injuries
(2000 IU and up)
4) Probiotics (Lacidophilus), which helps balance
immune function (approximately 15  live
organisms)
5) A phytonutrient green drink to help quench
damaging free radicals
“Somewhere, someone’s in training. And when you
meet him he will beat you” (Navy seals mantra).
   

A concussion is a traumatic brain injury that may result
in a bad headache, altered levels of alertness, or
unconsciousness. It temporarily interferes with the way
your brain works. Concussions are on the rise in high

SEPTEMBER 2013

school sports, and it can occur in any sport or reaction
activities. So, all coaches and parents need to have an
athlete evaluated when they have any kind of blow to
the head.

1) Continue with natural antiinflammatory
2) Continue with magnesium
3) Acetyl1carnitine: helps with brain function
The first priority nutritionally would be to help heal the
current injury – in this case, the part of the brain injured
by the concussion. By speeding the healing process,
the overall pain and the duration of pain is reduced,
thereby lessening the amount of substance P
(associated with inflammatory process and pain)
released within the thalamus of the brain. And also to
decrease the activation of the brain’s immune cells,
which are the source of inflammatory cytokines.
DHA, Zinc, Turmeric
Using these nutrition and nutritional supplements
during healing is going to enhance the healing response
and attempt to prevent adverse changes in the brain.
Reviewed studies reveal that the speedy intake of the
below macro and micro nutrients should be made
common practice “almost immediately” – both right
after a concussion and for two weeks following the
concussion.


1) Protein: Helps heel the injury. Take 1g/kg of body
weight, starting within a day of the injury
2) Creatine: Helps give the brain an intense and
immediate hit of energy needed to help cells heal
right after an injury
3) Reducing inflammatory damage to the brain:
a) DHA: an omega 3 fish oil, which is an
essential brain lipid that is critical for maximal
brain health and protection
b) Grape seed extract, bromelain, quercetin,
ginger
c) Polyphenols – turmeric, resveratrol
4) Antioxidants – alpha lipoic acid: Protects both the
fatty and water soluble part of the cells
5) Choline: Critical for brain development
6) Vitamin D: All the known benefits, and now
considered neuroprotective as well
7) Zinc: Enzyme for central nervous system (CNS)
health. The brain is a part of the CNS.
8) Magnesium: One of the best weapons against
delayed brain injury. Magnesium is one of these
incredible elements that play a role in a number of
biological processes. It is a cofactor in over 300
metabolic reactions, reduces inflammation, and
elevates glutathione in cells (the cell’s major
antioxidant). Low magnesium in the brain has also
been shown to greatly increase the vulnerability of
the brain to injury.
If symptoms persist past a reasonable amount of
healing time, then it is likely the brain (thalamus) is
struggling. This means the ratio of substance P to
BDNF (Brain Derived Neurotrophic Factor) is off.
Substance P is in excess and there is not enough BBNF.
SEPTEMBER 2013
A New York state concussion bill was signed on
September 2011. The legislation will prevent students
from returning to play until they have been without
symptoms for at least one day and have been cleared by
a physician.

         


        
        
          
       

        

        

          
      
      
         
       
     
      
        
        
          
      


         
     
      


           

         


            
          
           
       





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


Ascocid 1000
(Time-Disintegrating Vitamin C)
LYC 2000
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Cal & Mag Chelated Caps
(Fast-Dissolving Calcium and Magnesium Capsules)
Cal-C Caps and Cal-Cee Tablets
(Calcium Citrate Supplements)
Extress Super
(B-Complex 50s Formulation)
Lipo-Key
(A Combination Formulation for Liver Support)
Methyl-B12 Plus Oral Drops
(Methyl B12 with Folic Acid and B6 - Homocysteine)
Monolaurin 600 & 300
(Used to Retard Viral Invasion of the Body and Seasonal Allergies)
Opti-Plus
(Used for Healthy Eyes and Macular Degeneration)
Samolinic
(Norwegian Cold Water Salmon Oil with DHA and EPA)
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Please visit our website for details and complete
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Phone Orders: 800-325-9592 - 314-965-6699
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




This is the first in a series of articles that will take a
detailed look at the GMO issue otherwise known as
genetically modified organisms. Political and health
impacts of genetically modified foods or GMfoods,
will be explored in detail throughout this series. In all, I
anticipate that I will answer approximately 100
questions on this topic.
These articles represent my first draft writings that,
after continued development and expansion, will
appear in my upcoming book scheduled for release in
November 2013 entitled,  
       

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I think that it is appropriate to say that the GMO issue
will be around for a long, long time. Not all of the
evidence is in supporting either the safety or potential
dangers of GMfoods. I will say however, that I do not
believe that there is sufficient evidence to have placed
GMfoods on the market at this time. I will also go so
far as to say that I do not believe that GMfoods are
safe. I based my decision on extensive research into
both the political and scientific aspects of the GM
issue. Over the course of my continued articles you will
be left to make your own decisions. Maintaining an
open mind when reading my articles, or exploring any
other controversial topic that health care providers are
faced with, is not only crucial, but absolutely necessary
for objective, scientific exploration and advancement.
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
The term, “genetically modified organism” refers to an
organism whose genetic material has been modified
due to genetic engineering techniques. GM
technologies are generally used to alter the makeup of
organisms such as animals, plants, or bacteria. GMO’s
are the source of many modified foods and are used
widely in scientific research to produce other goods as
well. In 2006, specific countries that grew 97% of the
global crops with transgenic genes were the United
SEPTEMBER 2013
States (53%), Argentina (17%), Brazil (11%), Canada
(6%), India (4%), China (3%), Paraguay (2%), and
South Africa (1%). Within the next decade researchers
expect to see a drastic increase in the use of GMOs and
GM technologies in industrialized nations. You might
find it incredible to learn that limited studies have been
done demonstrating the safety of GM food
consumption by humans, and several of those studies
that have been conducted have shown serious health
problems including increased cancer risk, fertility
issues, inflammation in the intestines and other
potential health issues.
       

Monsanto Company, an American multinational
agricultural biotechnology corporation headquartered in
Missouri, is a leading producer of genetically
engineered seed. The company, founded in 1901 by
John Francis Queeny, operated primarily as a major
producer of plastics, including polystyrene and
synthetic fibers. As a chemical company, notable
achievements by Monsanto and its scientists included
being the first company to massproduce LEDs as well
as performing breakthrough research on catalytic
asymmetric hydrogenation. Multiple products
manufactured by the company deemed controversial
include insecticide DDT, PCBs (dielectric and coolant
fluids), and Agent Orange. Monsanto Company has not
been known for its honesty or transparency. A major lie
of Monsanto Company was that the safety and benefits
of the ingredients in GMOs were well established. In
fact, no longterm studies about either the safety or
benefits of GMO ingredients exist. The US Food and
Drug Administration do not currently require safety
studies of GMOs. Some independent studies currently
going on also raise questions as to allergies and other
health risks that could result from the consumption of
genetically modified food products.
In 1983, Monsanto, along with three academic teams,
were among the first to genetically modify a plant cell,
as well as conduct field trials of genetically modified
crops. The first genetically modified food was the
tomato. Between 1997 and 2002 the company came to
focus heavily on biotechnology after a process of
mergers and spinoffs that specified the company.
Monsanto pioneered the biotechnology industry by
applying its business model to agriculture. A “March
Against Monsanto” movement began earlier this year
to demonstrate in protest against GMOs and US
chemical giant Monsanto. Monsanto claims that there
are no dangers to the consumption of GMfoods by
people, but my articles will prove otherwise.
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

There are eight main steps involved in the development
of GM crops. Firstly, the gene of interest is isolated. It is
necessary to have existing knowledge about the
structure, location, and/or function of a gene on a
chromosome in order to identify the specific gene
responsible for a desired trait in an organism. Some
desired traits for GMO plants include tolerance to
drought or resistance to insects. After isolating the gene,
it is inserted into a transfer vector. Most commonly, a
plasmid, or circular model of DNA, is used to transfer
the isolated gene from the naturally occurring soil
bacterium. Using rDNA, the gene of interest is inserted
into the plasmid. The modified cell containing the
plasmid and the new gene is combined with cells from
the original plant, and some of the cells take up the
plasmid containing the transfer DNA.
After transformation, genes responsible for resistance are
inserted into the plasmid and are transferred along with
the genes containing the desired traits. When the cells
are exposed to an antibiotic, herbicide, etc., only the
cells that were transformed survive. The transformed
cells can then be regenerated to form entire plants by
culturing tissue. After GMO plants are grown, tests are
performed to determine the number of copies of the gene
inserted, whether or not the copies are intact, and
whether or not the gene is functional. Assessments of the
food and environmental safety of the GMOs are also
conducted in conjunction with testing of plant
performance.
There are concerns that GMfoods may cause antibiotic
resistance in people who consume the GM pesticide
containing foods. Other concerns involve contamination
of the environment with GMDNA (genetic material),
increased risk of various diseases including cancer and
gastrointestinal disease and autoimmune health
problems.
Subsequent articles in this series will be presented in this
identical question and answer format. Stay tuned for
much, much more on this controversial topic.

Dr. Michael Wald, nicknamed the Blood Detective for
his advanced clinical abilities and technological
developments, holds degrees as a doubleboard certified
nutritionist, a Certified DieticianNutritionist, a Certified
Nutritional Specialist and is a doctor of chiropractic and
earned his medical degree (MD) for educational
purposes from the University of Health Sciences School
of Medicine, Antigua. He is the director of nutrition at
Integrated Medicine of Mount Kisco, P.C. and has


Spring 2006
authored several books, hundreds of articles and has
provided continuing education for doctors across the
United States and abroad. He developed the
breakthrough software program for the nutritional
interpretation of laboratory work, that
has revolutionized clinical nutritional practice. He can
be reached at: www.intmedny.com,
www.blooddetective.com, www.zombiefoodbar.com or
[email protected].

1) http://www.ornl.gov/sci/techresources/Human_Genome/
elsi/gmfood.shtml
2) http://en.wikipedia.org/wiki/Monsanto
3) http://www.huffingtonpost.com/2013/05/25/march
againstmonsantogmoprotest_n_3336627.html
4) http://www.nepadbiosafety.net/subjects/biotechnology/
processofdevelopinggeneticallymodifiedgmcrops
5) http://recipes.howstuffworks.com/5commongenetically
modifiedfoods3.htm
6) http://shiftfrequency.com/comprehensivelistofgmo
products/
7) http://www.disabledworld.com/fitness/gmfoods.php
8) http://www.rumormillnews.com/cgibin/archive.cgi?
read=44805
9) http://en.wikipedia.org/wiki/tgenetically modified
food#Highly_processed_derivatives_containing_little_to
_no_DNA_or_protein
10) http://www.globalresearch.ca/potentialhealthhazards
ofgeneticallyengineeredfoods/8148
11) http://www.greenmedinfo.com/blog/alertgmocornand
roundupcausedcancerandkilledrats
12) http://www.reuters.com/article/2012/09/19/usgmcrops
safetyidUSBRE88I0L020120919
13) http://www.gmocompass.org/eng/grocery_shopping/
p r o cessed fo o d s/1 5 3 .ani mal feed ge ner ic
engineering.html
14) http://www.ucsusa.org/food_and_agriculture/ourfailing
foodsystem/industrialagriculture/theyeatwhatthe
realityof.html
15) http://enhs.umn.edu/current/5103/gm/harmful.html
16) h t t p : / / e a r t h o p e n s o u r c e . o r g / f i l e s / p d f s /
GMO_Myths_and_Truths/GMO Myths and Truths
1.3b.pdf
17) http://en.wikipedia.org/wiki/Intellectual_property
18) http://digital.law.washington.edu/dspacelaw/bitstream/
handle/1773.1/511/19PacRimL&PolyJ459010).pdf?
sequence=3
19) http://www.biogeneticservices.com/dnagmo.htm
20) http://www.brainwaving.com/2010/07/28/genetically
modifiedanimals/
21) http://www.asil.org/insigh38.cfm
22) h t t p : / / w w w . b e t t e r h e a l t h . v i c . g o v . a u / b h c v 2 /
bhcarticles.nsf/pages/Genetically_modified_foods
23) h t t p : / / a r t i c l e s . m e r c o l a . c o m / s i t e s / a r t i c l e s /
archives/2004/01/24/gmfoods.aspx
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SEPTEMBER 2013

IODORAL®
Lugol solution in tablet form
Available in tablets of 12.5 mg packaged
in bottles of 90 tablets and 180 tablets
Now available also in tablets of 50 mg
packaged in bottles of 30 and 90 tablets
Based on the collective experience of U.S. physicians who used Lugol solution extensively
in their practice for iodine supplementation over the past century, the recommended
daily intake for iodine supplementation was 0.1 to 0.3 ml containing 12.5 to 37.5
mg elemental iodine (1-3). We recently confirmed the keen observation of our medical
predecessors: this is exactly the range of iodine/iodide intake required for whole body
sufficiency, based on a recently developed iodine/iodide loading test (3). For non obese
subjects, whole body sufficiency for iodine can be achieved within 3 months with daily
intake of 12.5 to 50 mg (4,5).
1)
2)
3)
4)
5)
Abraham, G.E., Flechas, J.D., Hakala, J.C., Orthoiodosupplementation: Iodine sufficiency of the whole human body. The Original Internist, 9:30-41, 2002.
Gennaro, A.R., Remington: The Science and Practice of Pharmacy, 19th Edition, 1995, Mack Publishing Co., 976 & 1267.
Abraham, G.E., The safe and effective implementation of orthoiodosupplementation in medical practice. The Original Internist, 11:17-33, 2004.
Abraham, G.E., The concept of orthoiodosupplementation and its clinical implications. The Original Internist, 11:29-38, 2004.
Abraham, G.E., The historical background of the iodine project. The Original Internist, 12(2):57-66, 2005.
For further information on:
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IODORAL
Reprints of relevant articles
How to implement orthoiodosupplementation in your practice
How to request kits for the iodine/iodide loading test
Vist our website at www.optimox.com
Or contact us at: OPTIMOX CORPORATION P.O. Box 3378 Torrance, CA 90510-3378
or Call Toll Free: (800) 223-1601 or Fax: (310) 618-8748 or Email: [email protected]
: (
tremor, pallor, and perspiration. The migraine sufferer
may experience different identifiable migraines
(migraines that have no classification), and also
experience the tension type headaches as well.
The  is characterized by an
 or a "warning" symptom that may range from a
very few minutes to nearly an hour announcing the
expected arrival of an oncoming headache. Most
migraine sufferers report some sort of visual
disturbance as their aura. Flashing lights, intermittent
or zigzagging lines, a blind spot in the field of vision,
and photophobia are commonly reported. Several
sufferers also report a paresthesia or numbness.
Vomiting may provide relief. Even if the patient does
not vomit, most migraineurs report they think they
would feel better if they could. The "National Migraine
Foundation" reports that 70% of all migraine sufferers
are women, and 65% occur around the menstrual cycle.

 Allergies are common contributors to
migraines and should always be considered in addition
to spinal subluxations. The most commonly cited
suspected allergens include inhalants such as perfumes,
tobacco smoke and petroleum products. Foods that are
commonly reported for triggering migraines include,
but are certainly not limited to, chocolate, alcohol, all
dairy, several assorted nuts, beef, pork, caffeine, and
fish. All preservatives including glutamate, aspartame,
nitrates, monosodium and others should be suspect.
Aspartame is often cited to trigger allergic reactions.
: Several vitamin and mineral deficiencies
have been reported to be associated with various
  Consider
magnesium, B
complex vitamins, Omega3 Fatty Acids, and
tocopherols.
 Multichannel blood chemistries, CBC with
differential, urinalysis, circulation studies, and xrays to
the cervical and upper thoracic spine should all be
considered.
   This type headache is
characterized by severe pain (that may begin mild)
localized unilaterally in the temporal region with
localized scalp tenderness and signs of vascular
inflammation. The pain may be greatly increased by
laying down. Other symptoms may include depression,
gastrointestinal, fever, and night sweats. If the
inflammation is part of more widespread vascular
disorders the pain may well be bilateral. The arteries
most often affected are the superficial temporal,


Spring 2006
vertebral, ophthalmic, and the posterior ciliary arteries.
The internal and external and central retinal arteries are
less frequently affected. As temporal arteritis
progresses, red nodules may appear over the temporal
regions. Palpable tenderness may only be elicited over
the affected portion of the artery(ies). The affected
portion of the vessel palpates hard, convoluted, and
incompressible.
Blindness can result if the blood vessels serving
the optic nerve become involved. The symptoms of
ophthalmic artery ischemia are reported to begin with
diplopia or transient vision blurring especially upon
awakening. At the onset, an ophthalmic examination
may be normal but within two days papilledema,
hemorrhages and exudates can develop.
   An elevated ESR typically
above 4050 mm/hour and frequently above 90 mm/
hour. A negative ESR does not rule out temporal
arteritis. Giant cell arteritis confirmed by biopsy of the
affected artery provides the definitive diagnosis.
Multichannel blood chemistries, CBC with differential,
Doppler/plethysmography, and spirometry should be
considered. Temporal arteritis is a cardiovascular
condition and its underlying causes should be
addressed.

Drug induced headaches are referred to as rebound
headaches and are a result of regular use of pain
relieving medications. A truly vicious cycle usually
begins ominously by pain relievers taken even as little
as two to three days a week. Consequently, as soon as
the medicine wears off, hypersensitive receptors, in
susceptible individuals, turn on to produce a new
headache. That leads the headache sufferer to take
more medicine which in turn leads to more headaches.
Before long, most rebound patients are taking headache
medicine every day. Even overthecounter medications
can lead to rebound headaches (as well as the stronger
medications such as codeine, barbiturates and
ergotamine)      
        
    It is believed that
too much pain medication alters the serotonin levels so
the chemical loses its effectiveness. Be sure to look for
allergic reactions.




SEPTEMBER 2013
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

Dr. P. Reginald Hug*
Birmingham, AL

Dr. David Mulholland
Anchorage, AK
Dr. Debra Carpenter
Pueblo West, CO
Dr. Cheryl Vincent
Simsbury, CT
Dr. Thomas Jensen
Sterling, IL
Dr. Lynn Betz
Auburn, KS
Dr. Terry Collinson
Colorado Springs, CO

Dr. John Findlay
W. Palm Beach, FL
Dr. Theodore Johnson
Chicago, IL
Dr. Ben Bowers
Wichita, KS
Dr. James McGinn, Jr.
Crystal Lake, IL
Dr. Richard Brown
Olathe, KS
Dr. Michelle Oliver
Springfield, IL
Dr. Susan BuchananCheney
Phillipsburg, KS
Dr. Anthony Pantanella
Hoffman Estates, IL
Dr. Ralph Cardin
Overland Park, KS
Dr. Michael Poierier
Lombard, IL
Dr. H.M. Chalker
Meade, KS
Dr. Robert Pyne, Jr.
Palos Hills, IL
Dr. Dustin Cheney
Phillipsburg, KS
Dr. William Shelton
Lombard, IL
Dr. Rodney Clements
Eldorado, KS
Dr. Douglas Stam
Bourbonnais, IL
Dr. Paul Hughes
Edgerton, KS
Dr. Melanie Tiahrt
Alton, IL
Dr. Tobi Jeurink
Gardner, KS
Dr. Steven Zaeske
Orland Park, IL
Dr. Katherine Kubovy
Overland Park, KS
Dr. Alex Zevan, III
Bloomingdale, IL
Dr. Christena Nicholson
Overland Park, KS
Dr. Stephen Boudro
Elmhurst, IL

Dr. John Bernzott
Connersville, IN
Dr. Janie Pirner
Wichita, KS
Dr. Shawn M. Breton
Arlington Heights, IL
Dr. Thomas Jansen
Kendalville, IN
Dr. Rhonda Button
Carmi, IL
Dr. William Lyden
Mishawaka, IN
Dr. Christine Cosgrove
Roselle, IL
Dr. Brian McGuckin
Valparaiso, IN

Dr. Stephanie Clay
Baton Rouge, LA
Dr. Sharon DeFrain
Peotone, IL
Dr. Robert Prather
Indianapolis, IN
Dr. Robert W. Smith
Baton Rouge, LA
Dr. Mete Durum
Arlington Heights, IL

Dr. Ramneek Bhogal
Davenport, IA

Dr. Wayne Sodano
Bel Air, MD
Dr. Gary Bowden
McGregor, IA

Dr. Nancy Bronstein
Great Barrington, MA
Dr. Rita Cummings
Denver, CO
Dr. Stan Throckmorton
Anchorage, AK
Dr. Paula Dechert
Denver, CO

Dr. Michael Cessna
Tucson, AZ
Dr. Jeffrey Gappa
Brighton, CO
Dr. Laura Frey
Tucson, AZ
Dr. Timothy Gerhart
Glendale, AZ
Dr. Kellie Gray
Glendale, AZ
Dr. Michael Stone
Tucson, AZ


Dr. Lance Clouse
Van Buren, AR

Dr. Michael Brunner
Glendale, CA
Dr. Lewis Holm
Littleton, CO
Dr. William Kleber
Berthoud, CO
Dr. Reiner Kremer
Franktown, CO
Dr. Steven Lokken
Colorado Springs, CO
Dr. Duane Lowe
Colorado Springs, CO
Dr. Brandon Lundell
Longmont, CO
Dr. David Paradiso
Colorado Springs, CO
Dr. Christine Cohn
Newport Coast, CA
Dr. Corrie Pillon
Denver, CO
Dr. Ai Lien Diep
Los Angeles, CA
Dr. Philip Pollock
Sterling, CO
Dr. Jan Dooley
Arcata, CA
Dr. Deborah Riekman
Colorado Springs, CO
Dr. Jeffrey Greene
Los Angeles, CA
Dr. Kimberly Schmidt
Ft. Collins, CO
Dr. Valerie Johnson
Los Angeles, CA
Dr. Christie Sonchar
Colorado Springs, CO
Dr. Jill Jordan
Carlsbad, CA
Dr. Thomas Turner
Boulder, CO
Dr. Andrew Lucas
Riverside, CA
Dr. Michael Vanaria
Boulder, CO
Dr. Kathleen Power
Pasadena, CA
Dr. Brian Wilson
Englewood, CO
Dr. Rowen Richardson
Glendora, CA

Dr. Gina Carucci
Wethersfield, CT
Dr. Scott Soluk
Los Angeles, CA
Dr. Sylvie Wellhausen
Loma Linda, CA
Dr. Kelly Worth
Orange, CA

Dr. Robert Arne
Littleton, CO
Dr. John Baer
Englewood, CO
* Retired DABCI Practitioner
Dr. David Frerking
Tavares, FL
Dr. Marguerite Gerger
Clearwater, FL
Dr. Janice Piro
Palm Harbor, FL
Dr. Susan Player
Clearwater, FL
Dr. John Podlaski
Ocala, FL

Dr. Larry Haberski
Stone Mountain, GA

Dr. Uma Mulnick
McCall, ID

Dr. Delilah Anderson
Sandwich, IL
Dr. Jeffrey Bergin
Lindenhurst, IL
Dr. Rachael Fabbi
St. Charles, IL
Dr. Raymond Ferre
Decatur, IL
Dr. Mark Fredrick
Gurnee, IL
Dr. Suzanne Chester
Simsbury, CT
Dr. David Hepler
Lincoln, IL
Dr. Paul DiDomizio
Wolcott, CT
Dr. William Hogan
Lombard, IL
Dr. Ralph Manfredi
New Fairfield, CT
Dr. Lester Holze, Jr.
Elgin, IL
Dr. Mark Pappas
West Haven, CT
Dr. Cindy Howard
Orland Park, IL
Dr. Joanne Santiago
Avon, CT
Dr. Frederick Hult
McHenry, IL
Dr. Grant Iannelli
Lombard, IL
Dr. Darlene Ehlers
Tipton, IA
Dr. Robert Friedrichs
Mason City, IA
Dr. Alvin Schwerdtfager
Lindsborg, KS
Dr. Ron Young
Salina, KS

Dr. Daniel M. McGregor
Prudenville, MI

Dr. Tracy A. Stomgren
Glenwood, IA

Dr. Jeffrey Anderson
Edina, MN
Dr. Lynn Theesfield
Ames, IA
Dr. Robert Bergan
Minneapolis, MN
Dr. Zach Watkins
Johnston, IA
Dr. Timothy Bertsch
Champlin, MN
Dr. Anita Wubenna
Parkview, IA
Dr. Linda Bowers
Bloomington, MN

Dr. Mark Albers
Wichita, KS
Dr. Russell DesMarais
St. Paul, MN


Dr. Joel Eichers
Chanhassen, MN
Dr. Terry Nelson
Independence, MO
Dr. Sandrine Martin
Cornelius, NC
Dr. John Gerber
Blaine, MN
Dr. R Vincent Satterwhite
Kansas City, MO
Dr. Jacqueline McKool
Rutherfordton, NC
Dr. Timothy Gerhart
Red Wing, MN
Dr. Jeremy Thornton
Stockton, MO
Dr. Barbara Saunders
Garner, NC
Dr. Jedidiah Krauss
St. Louis Park, MN
Dr. Jeffrey S. Ware
Lake St. Louis, MO
Dr. Todd Smith
WinstonSalem, NC
Dr. Mac Beth Lindstrom
Slayton, MN
Dr. Robert Wiehe
West Plains, MO
Dr. Mark Yeager
Charlotte, NC
Dr. Todd McGillick
Gaylord, MN

Dr. Christopher Murray
Hastings, NE

Dr. Robert Gilbert
Mansfield, OH
Dr. Mallory Rupp
Grand Island, NE
Dr. Mark McAdoo
Athens, OH
Dr. Scott Sole
Kearney, NE
Dr. Van Merkle
Dayton, OH

Dr. Howard Balduc
Las Vegas, NV

Dr. Gerry Langston
Tulsa, OK
Dr. Craig Roles
Henderson, NV
Dr. Mark Mercer
Mannford, OK
Dr. Sandra Spore
Stillwater, MN

Dr. Jon Mastrobattista
Bernardville, NJ
Dr. Colleen Robinson
Duncan, OK
Dr. Leslie Stewart
St. Paul, MN
Dr. Perry Ricci
Egg Harbor City, NJ
Dr. Charles Strauman
St. Louis Park, MN

Dr. John Dalton
Roswell, NM
Dr. Thomas Miller
Coon Rapids, MN
Dr. Joseph Muldoon
Slayton, MN
Dr. Brenwyn Peddycoat
White Bear Lake, MN
Dr. Gregory Peterson
Winona, MN
Dr. Dane Roubos
Bloomington, MN
Dr. Terese Tomanek
Duluth, MN
Dr. Timothy Whelan
New Hope, MN
Dr. Jon Williams
Bloomington, MN

Dr. David Clark
Oak Grove, MO
Dr. Charles Eckert
Raymore, MO
Dr. Scott Hollis
Blue Springs, MO
Dr. Jay Kessinger
Rolla, MO
Dr. Darren Kirchner
Kahoka, MO
Dr. Kelley Kirchner
Kahoka, MO
Dr. Mable Leckrone
Liberty, MO
Dr. Duane Lowe
Maplewood, MO

Dr. John H. Gelhot
Albuquerque, NM

Dr. Shereen Jegtvig
Albuquerque, NM

Dr. Ronald Safko
New York City, NY
Dr. John Zilliox
Amherst, NY
Dr. Richard Santelli
Bethany, OK
Dr. Michael Taylor
Tulsa, OK


Dr. Daniel Beeson
Portland, OR
Dr. David Braman
Tuelatin, OR
Dr. Kathleen M. Galligan
Oregon City, OR
Dr. Edward M. Geller
Medford, OR
Dr. Usha Honeyman
Corvallis, OR
Dr. David Wickes
Portland, OR
Dr. Joe Lindley
Houston, TX

Dr. Bruce Fink
Coudersport, PA
Dr. Tim McCullough
Houston, TX

Dr. Mark Homison
Cranberry Township, PA
Dr. Karen L. Jorgensen
Pittsburgh, PA
Dr. John LaHoda
Richboro, PA
Dr. Fredrick Osterberg
Red Lion, PA

Dr. Jennifer Welch
Westerly, RI


Dr. Jon Bergrin
Florence, SC
Dr. Bruce Gwinnup
Charleston, SC
Dr. Peter Kfoury
Charleston, SC
Dr. Morgan Kutzner
Greenville, SC
Dr. Robert Pascal
Charleston, SC
Dr. Virginia Samuel
Columbia, SC

Dr. Roger Bommersbach
Brookings, SD
Dr. V.M. Thompsom
Arlington, TX


DR. Richard Allen
Sandy, UT
Dr. Don Vradenburg
St. George, UT

Dr. Robert Duca
Dunn Loring, VA
Dr. Guntrang Khalsa
Herndon, VA

Dr. H. Earl Moore
Spokane, WA

Dr. Michael Berglund
Kenosha, WI
Dr. Leslie Best
Madison, WI
Dr. Barbara Bradley
Wausau, WI
Dr. David Schwierert
Rapid City, SD
Dr. Bernie Finch
Pepin, WI

Dr. William Strauss
Lebanon, TN
Dr. Gwendolyn Gauerke
Iola, WI
Dr. Phillip Arnone
Matthews, NC
Dr. Kenzie Maloy
Hermiston, OR
Dr. Ralph Burton
Kennedale, TX
Dr. Stephen Button
Mount Airy, NC
Dr. Scott Northrup
Brookings, OR
Dr. Lance CarltonDurrett
The Woodlands, TX
Dr. Karen Carrick
Raleigh, NC
Dr. Kristopher Peterson
Hermiston, OR
Dr. Victor Carsrud
Austin, TX
Dr. Rick Davis
Conover, NC
Dr. Thomas Richards
Beaverton, OR
Dr. Janie Duke
Plano, TX
Dr. Nikolas R. Hedberg
Asheville, NC
Dr. James Siegel
Canyonville, OR
Dr. Steve Grimm
San Antonio, TX
Dr. Dean Kenny
High Point, NC
Dr. Mark Thomas
Cottage Grove, OR
Dr. Doreen LewisOverstrom
San Antonio, TX
Spring 2006
Dr. Mike Prioux
Friendswood, TX
Dr. Kevin Branham
Eagle River, WI
Dr. Steven Lumsden
Gresham, OR


Dr. Gregory Mrozinski
Houston, TX
Dr. Roger Prill
Mitchell, SD

Dr. Edward Brown
Dallas, TX

Dr. William R. Armstrong
Laurenburg, NC
Dr. Zachery McVey
League City, TX
Dr. Craig Gilbaugh
Ashland, WI
Dr. Kathleen Maedke
Milwaukee, WI
Dr. Cheryl Metzler
Green Bay, WI
Dr. Gina R. Schultz
Blanchardville, WI
Dr. David A. Sommerfield
Rice Lake, WI
Dr. Dean Willhite
Manitowoc, WI
Dr. Kelly G. Worth
Racine, WI
SEPTEMBER 2013
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

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


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